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10.3389/fmed.2015.00060 http://scihub22266oqcxt.onion/10.3389/fmed.2015.00060 C4553389!4553389!26380262     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Med+(Lausanne) 2015 ; 2 (ä): ä Nephropedia Template TP {{tp|p=26380262|t=2015. Chronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solutes |pdf=|usr=}}{{26380262}} gab.com Text Chronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solutes JO: Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=26380262 #FOAvip Chronic kidney disease (CKD) is a major global health concern, and the uremic state is highly associated with fibrogenesis in several organs and tissues. Fibrosis is characterized by excessive production and deposition of extracellular matrix proteins with a detrimental impact on organ function. Another key feature of CKD is the retention and subsequent accumulation of solutes that are normally cleared by the healthy kidney. Several of these uremic retention solutes, including indoxyl sulfate and p-cresyl sulfate, have been suggested to be CKD-specific triggers for the development and perpetuation of fibrosis. The purpose of this brief review is to gather and discuss the current body of evidence linking uremic retention solutes to the fibrotic response during CKD, with a special emphasis on the pathophysiological mechanisms in the kidney. Twit Text FOAVip Chronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solutes ????Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=26380262 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26380262 #FOAvip English Wikipedia <ref>{{Cite pmid|26380262}}</ref> Chronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solutes #MMPMID26380262 Mutsaers HAM; Stribos EGD; Glorieux G; Vanholder R; Olinga P Front Med (Lausanne) 2015[]; 2 (ä): ä PMID26380262show ga Chronic kidney disease (CKD) is a major global health concern, and the uremic state is highly associated with fibrogenesis in several organs and tissues. Fibrosis is characterized by excessive production and deposition of extracellular matrix proteins with a detrimental impact on organ function. Another key feature of CKD is the retention and subsequent accumulation of solutes that are normally cleared by the healthy kidney. Several of these uremic retention solutes, including indoxyl sulfate and p-cresyl sulfate, have been suggested to be CKD-specific triggers for the development and perpetuation of fibrosis. The purpose of this brief review is to gather and discuss the current body of evidence linking uremic retention solutes to the fibrotic response during CKD, with a special emphasis on the pathophysiological mechanisms in the kidney. äChronic Kidney Disease and Fibrosis: The Role of Uremic Retention Solu(epmc).pdf DeepDyve Pubget Overpricing