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10.1155/2015/324173

http://scihub22266oqcxt.onion/10.1155/2015/324173
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C4549487!4549487!26347836
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suck abstract from ncbi


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pmid26347836      Case+Rep+Obstet+Gynecol 2015 ; 2015 (ä): ä
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  • Vitamin K Deficiency Embryopathy from Hyperemesis Gravidarum #MMPMID26347836
  • Lane AS; Stallworth JL; Eichelberger KY; Trofatter KF
  • Case Rep Obstet Gynecol 2015[]; 2015 (ä): ä PMID26347836show ga
  • A 21-year-old primigravida had a pregnancy complicated by hyperemesis gravidarum (HG) beginning at 7-week gestation. Despite medical therapy, she lost 18% of her prepregnancy weight. Early ultrasound at 14 weeks demonstrated a flattened facial profile with nasal hypoplasia (Binder phenotype) consistent with vitamin K deficiency from HG. She had a percutaneous endoscopic gastrojejunostomy tube placed for enteral feeding at 15-week gestation. At repeated anatomy ultrasound at 21-week gestation, delivery, and postnatal pediatric genetics exam, nasal hypoplasia was consistent with vitamin K deficiency embryopathy from HG. Nausea and vomiting of pregnancy is a common condition. HG, the most severe form, has many maternal and fetal effects. Evaluation of vitamin K status could potentially prevent this rare and disfiguring embryopathy.
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