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10.1002/mus.24619 http://scihub22266oqcxt.onion/10.1002/mus.24619 C4545465!4545465!25703692     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Muscle+Nerve 2015 ; 52 (5): 852-8 Nephropedia Template TP {{tp|p=25703692|t=2015. Amelioration of ischemia-reperfusion induced muscle injury by the recombinant human MG53 protein |pdf=|usr=}}{{25703692}} gab.com Text Amelioration of ischemia-reperfusion induced muscle injury by the recombinant human MG53 protein JO: Muscle Nerve http://www.kidney.de/pget.php?&tw=1&pmid=25703692 #FOAvip Introduction: Ischemia-reperfusion injury (I-R) in skeletal muscle requires timely treatment. Methods: Rodent models of I-R injury were used to test the efficacy of recombinant human MG53 (rhMG53) protein for protecting skeletal muscle. Results: In a mouse I-R injury model, we found that mg53?/? mice are more susceptible to I-R injury. rhMG53 applied intravenously to the wild type mice protected I-R injured muscle, as demonstrated by reduced CK release and Evans blue staining. Histochemical studies confirmed beneficial effects of rhMG53. Interestingly, rhMG53 did not protect against I-R injury in rat skeletal muscle. This was likely due to the fact that the plasma level of endogenous MG53 protein is high in rats. Discussion: Our data suggest that rhMG53 may be a potential therapy for protection against muscle trauma. A mouse model appears to be a better choice than a rat model for evaluating potential treatments for protecting skeletal muscle. Twit Text FOAVip Amelioration of ischemia-reperfusion induced muscle injury by the recombinant human MG53 protein ????Muscle Nerve http://www.kidney.de/pget.php?&tw=1&pmid=25703692 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25703692 #FOAvip English Wikipedia <ref>{{Cite pmid|25703692}}</ref> Amelioration of ischemia-reperfusion induced muscle injury by the recombinant human MG53 protein #MMPMID25703692 Zhu H; Hou J; Roe JL; Park KH; Tan T; Zheng Y; Li L; Zhang C; Liu J; Liu Z; Ma J; Walters TJ Muscle Nerve 2015[Nov]; 52 (5): 852-8 PMID25703692show ga Introduction: Ischemia-reperfusion injury (I-R) in skeletal muscle requires timely treatment. Methods: Rodent models of I-R injury were used to test the efficacy of recombinant human MG53 (rhMG53) protein for protecting skeletal muscle. Results: In a mouse I-R injury model, we found that mg53?/? mice are more susceptible to I-R injury. rhMG53 applied intravenously to the wild type mice protected I-R injured muscle, as demonstrated by reduced CK release and Evans blue staining. Histochemical studies confirmed beneficial effects of rhMG53. Interestingly, rhMG53 did not protect against I-R injury in rat skeletal muscle. This was likely due to the fact that the plasma level of endogenous MG53 protein is high in rats. Discussion: Our data suggest that rhMG53 may be a potential therapy for protection against muscle trauma. A mouse model appears to be a better choice than a rat model for evaluating potential treatments for protecting skeletal muscle. äAmelioration of ischemia-reperfusion induced muscle injury by the reco(epmc).pdf DeepDyve Pubget Overpricing