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10.3389/fendo.2015.00126 http://scihub22266oqcxt.onion/10.3389/fendo.2015.00126 C4538298!4538298!26347711     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Endocrinol+(Lausanne) 2015 ; 6 (ä): ä Nephropedia Template TP {{tp|p=26347711|t=2015. Pheochromocytomas and Paragangliomas: Clinical and Genetic Approaches |pdf=|usr=}}{{26347711}} gab.com Text Pheochromocytomas and Paragangliomas: Clinical and Genetic Approaches JO: Front Endocrinol (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=26347711 #FOAvip Pheochromocytomas (PCCs) and paragangliomas (PGLs) are neuroendocrine tumors derived from the chromaffin tissue. Diagnosis of these tumors is extremely important as they are linked to the hypertension syndrome with great cardiovascular morbidity and mortality. A great majority of PCCs and PGLs are sporadic and benign tumors; however, the classic idea of 10% exception of these features is changing. The description of new genes linked to familial forms of PCC/PGLs, such as succinate dehydrogenase (SDH) complex subunits, KIF1B?, EGLN1, TMEM127, and MAX, added to the well-known PCC familial syndrome (MEN2, VHL, and neurofibromatosis type 1) presents new challenges for diagnosis. In this review, we discuss the diversity of clinical and genetic approaches to this syndrome as well the diverse criteria that should guide genetic investigation. Twit Text FOAVip Pheochromocytomas and Paragangliomas: Clinical and Genetic Approaches ????Front Endocrinol (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=26347711 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26347711 #FOAvip English Wikipedia <ref>{{Cite pmid|26347711}}</ref> Pheochromocytomas and Paragangliomas: Clinical and Genetic Approaches #MMPMID26347711 Costa MHS; Ortiga-Carvalho TM; Violante AD; Vaisman M Front Endocrinol (Lausanne) 2015[]; 6 (ä): ä PMID26347711show ga Pheochromocytomas (PCCs) and paragangliomas (PGLs) are neuroendocrine tumors derived from the chromaffin tissue. Diagnosis of these tumors is extremely important as they are linked to the hypertension syndrome with great cardiovascular morbidity and mortality. A great majority of PCCs and PGLs are sporadic and benign tumors; however, the classic idea of 10% exception of these features is changing. The description of new genes linked to familial forms of PCC/PGLs, such as succinate dehydrogenase (SDH) complex subunits, KIF1B?, EGLN1, TMEM127, and MAX, added to the well-known PCC familial syndrome (MEN2, VHL, and neurofibromatosis type 1) presents new challenges for diagnosis. In this review, we discuss the diversity of clinical and genetic approaches to this syndrome as well the diverse criteria that should guide genetic investigation. äPheochromocytomas and Paragangliomas: Clinical and Genetic Approaches (epmc).pdf DeepDyve Pubget Overpricing