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10.1055/s-0034-1387924 http://scihub22266oqcxt.onion/10.1055/s-0034-1387924 C4535344!4535344 !25173498     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25173498 &cmd=llinks): Failed to open stream: HTTP request failed! 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Are microparticles the missing link between thrombosis and autoimmune diseases? Involvement in selected rheumatologic diseases |pdf=|usr=}}{{25173498 }} gab.com Text Are microparticles the missing link between thrombosis and autoimmune diseases Involvement in selected rheumatologic diseases JO: Semin Thromb Hemost http://www.kidney.de/pget.php?&tw=1&pmid=25173498 #FOAvip Microparticles (MPs) are membrane-bound vesicles with important physiologic effects. MPs exchange information intercellularly, with each kind of MP carrying antigens and receptors of the cells from which they originated. They are biologic effectors in inflammation, angiogenesis, vascular injury, and thrombosis. Thrombosis is generally caused by abnormalities in blood flow, blood composition, and/or properties of the vessel wall. Thrombosis is a well-described feature of cardiovascular disease and cerebrovascular disease. Accumulating evidence suggests that increased risk of thrombosis is also characteristic of autoimmune disorders and immune-mediated diseases affecting all age groups, although the older adults are most vulnerable. Current research has also implicated MPs as a source of autoantigenic nuclear material that can form immune complexes, activate the innate immune system, and may lead to autoimmunity. This review focuses on the contribution of MPs to both the pathogenesis of autoimmune diseases and, as the immune and coagulation systems are tightly linked, their role in hypercoagulability in the setting of autoimmunity in an aging population. Twit Text FOAVip Are microparticles the missing link between thrombosis and autoimmune diseases Involvement in selected rheumatologic diseases ????Semin Thromb Hemost http://www.kidney.de/pget.php?&tw=1&pmid=25173498 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25173498 #FOAvip English Wikipedia <ref>{{Cite pmid|25173498 }}</ref> Are microparticles the missing link between thrombosis and autoimmune diseases? Involvement in selected rheumatologic diseases #MMPMID25173498 Cunningham M ; Marks N ; Barnado A ; Wirth JR ; Gilkeson G ; Markiewicz M Semin Thromb Hemost 2014[Sep]; 40 (6 ): 675-81 PMID25173498 show ga Microparticles (MPs) are membrane-bound vesicles with important physiologic effects. MPs exchange information intercellularly, with each kind of MP carrying antigens and receptors of the cells from which they originated. They are biologic effectors in inflammation, angiogenesis, vascular injury, and thrombosis. Thrombosis is generally caused by abnormalities in blood flow, blood composition, and/or properties of the vessel wall. Thrombosis is a well-described feature of cardiovascular disease and cerebrovascular disease. Accumulating evidence suggests that increased risk of thrombosis is also characteristic of autoimmune disorders and immune-mediated diseases affecting all age groups, although the older adults are most vulnerable. Current research has also implicated MPs as a source of autoantigenic nuclear material that can form immune complexes, activate the innate immune system, and may lead to autoimmunity. This review focuses on the contribution of MPs to both the pathogenesis of autoimmune diseases and, as the immune and coagulation systems are tightly linked, their role in hypercoagulability in the setting of autoimmunity in an aging population. |Age Factors [MESH] |Animals [MESH] |Arthritis, Rheumatoid/immunology/*metabolism [MESH] |Autoimmune Diseases/immunology/metabolism [MESH] |Cell-Derived Microparticles/immunology/*metabolism [MESH] |Humans [MESH] |Lupus Erythematosus, Systemic/immunology/*metabolism [MESH] |Risk Factors [MESH]Are microparticles the missing link between thrombosis and autoimmune (epmc).pdf DeepDyve Pubget Overpricing