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10.1111/prd.12096 http://scihub22266oqcxt.onion/10.1111/prd.12096 C4530502!4530502 !26252408     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26252408 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 243.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26252408 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Periodontol+2000 2015 ; 69 (1 ): 160-80 Nephropedia Template TP {{tp|p=26252408 |t=2015. Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease |pdf=|usr=}}{{26252408 }} gab.com Text Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease JO: Periodontol 2000 http://www.kidney.de/pget.php?&tw=1&pmid=26252408 #FOAvip Dendritic cells are potent antigen-capture and antigen-presenting cells that play a key role in the initiation and regulation of the adaptive immune response. This process of immune homeostasis, as maintained by dendritic cells, is susceptible to dysregulation by certain pathogens during chronic infections. Such dysregulation may lead to disease perpetuation with potentially severe systemic consequences. Here we discuss in detail how intracellular pathogens exploit dendritic cells and escape degradation by altering or evading autophagy. This novel mechanism explains, in part, the chronic, persistent nature observed in several immuno-inflammatory diseases, including periodontal disease. We also propose a hypothetical model of the plausible role of autophagy in the context of periodontal disease. Promotion of autophagy may open new therapeutic strategies in the search of a 'cure' for periodontal disease in humans. Twit Text FOAVip Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease ????Periodontol 2000 http://www.kidney.de/pget.php?&tw=1&pmid=26252408 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26252408 #FOAvip English Wikipedia <ref>{{Cite pmid|26252408 }}</ref> Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease #MMPMID26252408 El-Awady AR ; Arce RM ; Cutler CW Periodontol 2000 2015[Oct]; 69 (1 ): 160-80 PMID26252408 show ga Dendritic cells are potent antigen-capture and antigen-presenting cells that play a key role in the initiation and regulation of the adaptive immune response. This process of immune homeostasis, as maintained by dendritic cells, is susceptible to dysregulation by certain pathogens during chronic infections. Such dysregulation may lead to disease perpetuation with potentially severe systemic consequences. Here we discuss in detail how intracellular pathogens exploit dendritic cells and escape degradation by altering or evading autophagy. This novel mechanism explains, in part, the chronic, persistent nature observed in several immuno-inflammatory diseases, including periodontal disease. We also propose a hypothetical model of the plausible role of autophagy in the context of periodontal disease. Promotion of autophagy may open new therapeutic strategies in the search of a 'cure' for periodontal disease in humans. |*Adaptive Immunity [MESH] |*Autophagy [MESH] |Dendritic Cells/*immunology/pathology [MESH] |Host-Pathogen Interactions [MESH] |Humans [MESH] |Mucous Membrane/immunology [MESH] |Periodontitis/*immunology/*microbiology/pathology [MESH]Dendritic cells: microbial clearance via autophagy and potential immun(epmc).pdf DeepDyve Pubget Overpricing