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10.14218/JCTH.2013.008XX http://scihub22266oqcxt.onion/10.14218/JCTH.2013.008XX C4521280!4521280!26357611     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Transl+Hepatol 2013 ; 1 (2): 138-43 Nephropedia Template TP {{tp|p=26357611|t=2013. Environmental Exposures and Hepatocellular Carcinoma |pdf=|usr=}}{{26357611}} gab.com Text Environmental Exposures and Hepatocellular Carcinoma JO: J Clin Transl Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=26357611 #FOAvip Infection with hepatitis B and/or hepatitis C virus is a well-established risk factor for the development of hepatocellular carcinoma (HCC). However, it is now clear that certain occupational, environmental, and lifestyle factors also play a role in cancer development. Among these factors are smoking, alcohol consumption, workplace exposure to vinyl chloride, and exposure to polycylic aromatic hydrocarbons and aflatoxins. There is also evidence that several other chemical and infectious agents have a role in inducing HCC in humans. Epidemiologic studies and the use of biomarkers have provided essential data to demonstrate the importance of some of these factors in human risk, while animal studies have suggested that other chemicals may also play a role. Although immunization against hepatitis B virus infection remains the primary method of preventing HCC in regions of the world where this virus is a primary etiologic agent, there is currently no vaccine for hepatitis C virus. Thus, limiting exposure to other known risk factors remains an important mechanism in preventing HCC. Twit Text FOAVip Environmental Exposures and Hepatocellular Carcinoma ????J Clin Transl Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=26357611 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26357611 #FOAvip English Wikipedia <ref>{{Cite pmid|26357611}}</ref> Environmental Exposures and Hepatocellular Carcinoma #MMPMID26357611 Santella RM; Wu HC J Clin Transl Hepatol 2013[Dec]; 1 (2): 138-43 PMID26357611show ga Infection with hepatitis B and/or hepatitis C virus is a well-established risk factor for the development of hepatocellular carcinoma (HCC). However, it is now clear that certain occupational, environmental, and lifestyle factors also play a role in cancer development. Among these factors are smoking, alcohol consumption, workplace exposure to vinyl chloride, and exposure to polycylic aromatic hydrocarbons and aflatoxins. There is also evidence that several other chemical and infectious agents have a role in inducing HCC in humans. Epidemiologic studies and the use of biomarkers have provided essential data to demonstrate the importance of some of these factors in human risk, while animal studies have suggested that other chemicals may also play a role. Although immunization against hepatitis B virus infection remains the primary method of preventing HCC in regions of the world where this virus is a primary etiologic agent, there is currently no vaccine for hepatitis C virus. Thus, limiting exposure to other known risk factors remains an important mechanism in preventing HCC. äEnvironmental Exposures and Hepatocellular Carcinoma (epmc).pdf DeepDyve Pubget Overpricing