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10.1016/j.bbamem.2015.01.013

http://scihub22266oqcxt.onion/10.1016/j.bbamem.2015.01.013
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C4520786!4520786!25644871
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suck abstract from ncbi


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pmid25644871      Biochim+Biophys+Acta 2015 ; 1848 (11 0 0): 3021-5
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  • S Typhimurium strategies to resist killing by cationic antimicrobial peptides #MMPMID25644871
  • Matamouros S; Miller SI
  • Biochim Biophys Acta 2015[Nov]; 1848 (11 0 0): 3021-5 PMID25644871show ga
  • S.Typhimurium is a broad host range Gram-negative patho that must evade killing by host innate immune systems to colonize, replicate, cause disease, and be transmitted to other hosts. A major pathogenic strategy of Salmonellae is entrance, survival, and replication within eukaryotic cell phagocytic vacuoles. These phagocytic vacuoles and gastrointestinal mucosal surfaces contain multiple cationic antimicrobial peptides (CAMPs) which control invading bacteria. S. Typhimurium possesses several key mechanisms to resist killing by CAMPs which involve sensing CAMPs and membrane damage to activate signaling cascades that result in remodeling of the bacterial envelope to reduce its overall negative charge with an increase in hydrophobicity to decrease binding and effectiveness of CAMPs. Moreover Salmonellae have additional mechanisms to resist killing by CAMPs including an outer membrane protease which targets cationic peptides at the surface, and specific efflux pumps which protect the inner membrane from damage.
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