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10.1016/j.smim.2015.03.007 http://scihub22266oqcxt.onion/10.1016/j.smim.2015.03.007 C4515383!4515383!25818531     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Immunol 2015 ; 27 (3): 177-83 Nephropedia Template TP {{tp|p=25818531|t=2015. Neutrophils and Inflammatory Resolution in the Mucosa |pdf=|usr=}}{{25818531}} gab.com Text Neutrophils and Inflammatory Resolution in the Mucosa JO: Semin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=25818531 #FOAvip Inflammatory diseases in mucosal organs as diverse as the lung, liver and intestine inevitably require the intimate interactions between neutrophils and epithelia. The physiologic consequences of such interactions often determine endpoint organ function, and for this reason, much recent interest has developed in identifying mechanisms and novel targets to promote the resolution of mucosal inflammation. Physiologically-relevant in vitro and in vivo model systems have aided in discovery of novel pathways to define basic inflammatory mechanisms and approaches to defining the concepts of inflammatory resolution. Here, we will review the recent literature regarding the contribution of neutrophils to inflammatory resolution, with an emphasis on the role of the tissue microenvironment, endogenous pathways for promoting resolution and the molecular determinants of neutrophil-epithelial cell interactions during ongoing inflammation. These recent studies highlight the dynamic nature of pro-resolving pathways and lend insight into the complexity of treating mucosal inflammation. Twit Text FOAVip Neutrophils and Inflammatory Resolution in the Mucosa ????Semin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=25818531 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25818531 #FOAvip English Wikipedia <ref>{{Cite pmid|25818531}}</ref> Neutrophils and Inflammatory Resolution in the Mucosa #MMPMID25818531 Colgan SP Semin Immunol 2015[May]; 27 (3): 177-83 PMID25818531show ga Inflammatory diseases in mucosal organs as diverse as the lung, liver and intestine inevitably require the intimate interactions between neutrophils and epithelia. The physiologic consequences of such interactions often determine endpoint organ function, and for this reason, much recent interest has developed in identifying mechanisms and novel targets to promote the resolution of mucosal inflammation. Physiologically-relevant in vitro and in vivo model systems have aided in discovery of novel pathways to define basic inflammatory mechanisms and approaches to defining the concepts of inflammatory resolution. Here, we will review the recent literature regarding the contribution of neutrophils to inflammatory resolution, with an emphasis on the role of the tissue microenvironment, endogenous pathways for promoting resolution and the molecular determinants of neutrophil-epithelial cell interactions during ongoing inflammation. These recent studies highlight the dynamic nature of pro-resolving pathways and lend insight into the complexity of treating mucosal inflammation. äNeutrophils and Inflammatory Resolution in the Mucosa (epmc).pdf DeepDyve Pubget Overpricing