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http://scihub22266oqcxt.onion/ C4515335!4515335!26221157     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Iran+J+Child+Neurol 2015 ; 9 (2): 9-16 Nephropedia Template TP {{tp|p=26221157|t=2015. Animal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn |pdf=|usr=}}{{26221157}} gab.com Text Animal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn JO: Iran J Child Neurol http://www.kidney.de/pget.php?&tw=1&pmid=26221157 #FOAvip Objective: Hypoxic insults are implicated in the spectrum of fetal disorders, including cerebral palsy (CP). In view of the major contribution of intrapartum risk factors and prematurity to subsequent neurological morbidity and mortality in humans, this study aimed to clarify the pathophysiology of brain injury, especiallyperiventricular white matter damage (WMD), that occur in utero to the immature and near-term fetal CNS. Materials & Methods: An evaluation of the resulting neurological and behavioural phenotype in the newborn was performed by utilising a battery of neurobehavioural tests, including the Morris water-maze and the open-field test, followed by cerebral MRI and histopathology. Results: This study used a murine model to examine the deleterious effects of WMD brought about by cerebral hypoxia-ischemia (HI) and the characteristic features of CP in mice. Murine models have proven themselves valuable in the area of experimental neuroscience. Conclusion: Hypoxia-treated mice were observed to demonstrate a significant neurofunctional deficit compared with sham mice on two behavioral measures. Indeed, different brain regions, including the sensorimotor cortex, the striatum, and the hippocampus were noticeably damaged after HI insult, as determined by bothMRI and histopathology. These results, albeit qualitative in nature, appear to support the pre-existing finding that the long-term neurofunctional outcome in animal subjects with CP is strongly associated with the anatomical extent and pattern of cerebral damage as determined by both delayed neuroimaging andhistopathology. Twit Text FOAVip Animal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn ????Iran J Child Neurol http://www.kidney.de/pget.php?&tw=1&pmid=26221157 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26221157 #FOAvip English Wikipedia <ref>{{Cite pmid|26221157}}</ref> Animal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn #MMPMID26221157 WILSON MD Iran J Child Neurol 2015[Spr]; 9 (2): 9-16 PMID26221157show ga Objective: Hypoxic insults are implicated in the spectrum of fetal disorders, including cerebral palsy (CP). In view of the major contribution of intrapartum risk factors and prematurity to subsequent neurological morbidity and mortality in humans, this study aimed to clarify the pathophysiology of brain injury, especiallyperiventricular white matter damage (WMD), that occur in utero to the immature and near-term fetal CNS. Materials & Methods: An evaluation of the resulting neurological and behavioural phenotype in the newborn was performed by utilising a battery of neurobehavioural tests, including the Morris water-maze and the open-field test, followed by cerebral MRI and histopathology. Results: This study used a murine model to examine the deleterious effects of WMD brought about by cerebral hypoxia-ischemia (HI) and the characteristic features of CP in mice. Murine models have proven themselves valuable in the area of experimental neuroscience. Conclusion: Hypoxia-treated mice were observed to demonstrate a significant neurofunctional deficit compared with sham mice on two behavioral measures. Indeed, different brain regions, including the sensorimotor cortex, the striatum, and the hippocampus were noticeably damaged after HI insult, as determined by bothMRI and histopathology. These results, albeit qualitative in nature, appear to support the pre-existing finding that the long-term neurofunctional outcome in animal subjects with CP is strongly associated with the anatomical extent and pattern of cerebral damage as determined by both delayed neuroimaging andhistopathology. äAnimal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn (epmc).pdf DeepDyve Pubget Overpricing