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10.1097/MOG.0000000000000119 http://scihub22266oqcxt.onion/10.1097/MOG.0000000000000119 C4512208!4512208!25255234     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25255234&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Opin+Gastroenterol 2014 ; 30 (6): 531-8 Nephropedia Template TP {{tp|p=25255234|t=2014. Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosis |pdf=|usr=}}{{25255234}} gab.com Text Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosis JO: Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=25255234 #FOAvip Purpose of review: Intestinal fibrosis is a serious, yet common, outcome in patients with inflammatory bowel disease (IBD). Despite advances in developing novel treatment modalities to control chronic gut inflammation characteristic of IBD, no effective anti-fibrotic therapies exist to date. As such, a deeper understanding of the molecular mechanisms underlying intestinal fibrosis and the availability of relevant animal models are critical to move this area of investigation forward. Recent findings: Emerging concepts in the pathogenesis of intestinal fibrosis include the central role of interleukin (IL)-17 and Th17 immune responses, although their precise contribution to chronic inflammation and IBD remains controversial. Other novel mediators of intestinal fibrosis, such as tumor necrosis factor (TNF)-like ligand 1A (TL1A) and components of the renin-angiotensin system, support the importance of IL-17. Additionally, recent studies utilizing novel mouse models highlight the significance of the gut microbiota and link components of bacterial sensing, including nucleotide-binding oligomerization domain-containing protein 2 (NOD2), to IL-17/Th17 immune responses in the development of inflammation-associated intestinal fibrosis. Summary: Recent progress in identifying key mediators, novel animal models, and important mechanistic pathways in the pathogenesis of intestinal fibrosis hold promise for the development of effective anti-fibrotics in an area of significant, unmet clinical need. Twit Text FOAVip Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosis ????Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=25255234 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25255234 #FOAvip English Wikipedia <ref>{{Cite pmid|25255234}}</ref> Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosis #MMPMID25255234 Ray S; De Salvo C; Pizarro TT Curr Opin Gastroenterol 2014[Nov]; 30 (6): 531-8 PMID25255234show ga Purpose of review: Intestinal fibrosis is a serious, yet common, outcome in patients with inflammatory bowel disease (IBD). Despite advances in developing novel treatment modalities to control chronic gut inflammation characteristic of IBD, no effective anti-fibrotic therapies exist to date. As such, a deeper understanding of the molecular mechanisms underlying intestinal fibrosis and the availability of relevant animal models are critical to move this area of investigation forward. Recent findings: Emerging concepts in the pathogenesis of intestinal fibrosis include the central role of interleukin (IL)-17 and Th17 immune responses, although their precise contribution to chronic inflammation and IBD remains controversial. Other novel mediators of intestinal fibrosis, such as tumor necrosis factor (TNF)-like ligand 1A (TL1A) and components of the renin-angiotensin system, support the importance of IL-17. Additionally, recent studies utilizing novel mouse models highlight the significance of the gut microbiota and link components of bacterial sensing, including nucleotide-binding oligomerization domain-containing protein 2 (NOD2), to IL-17/Th17 immune responses in the development of inflammation-associated intestinal fibrosis. Summary: Recent progress in identifying key mediators, novel animal models, and important mechanistic pathways in the pathogenesis of intestinal fibrosis hold promise for the development of effective anti-fibrotics in an area of significant, unmet clinical need. äCentral role of IL-17/Th17 immune responses and the gut microbiota in (epmc).pdf DeepDyve Pubget Overpricing