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http://scihub22266oqcxt.onion/ C4509175!4509175!26221230     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Clin+Exp+Med 2015 ; 8 (5): 6937-46 Nephropedia Template TP {{tp|p=26221230|t=2015. Platelet mitochondrial dysfunction of DM rats and DM patients |pdf=|usr=}}{{26221230}} gab.com Text Platelet mitochondrial dysfunction of DM rats and DM patients JO: Int J Clin Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=26221230 #FOAvip It is known that cardiovascular complications plays important roles in the development of diabetes mellitus (DM) and platelet dysfunction is one of the key reasons which led to microangiopathy. This study was designed to investigate the mitochondria function changes of platelet in DM rats and DM patients. The results showed that the platelets viability, platelet adenosine triphosphate (ATP) content and platelet mitochondrial ATP content of DM rats were lower than that of normal rats; when incubated in vitro for 24 h, platelet number and mitochondrial membrane potential (MMP) of DM rats were lower than that of normal rats, reactive oxygen species (ROS) was higher than that of normal rats. For DM patients, their platelet number and ROS were higher and MMP was lower than those of normal people; when incubated in vitro for 24 h, platelet viability of DM patients was lower than that of normal people. Platelet ultra-microstructures of DM rats and DM patients were abnormal. These results suggested that platelet mitochondrial function of both DM rats and DM patients was impaired when compared to normal rats and normal people, respectively. Platelets may be applied as a biomarker to observe the mitochondrial changes during DM. Twit Text FOAVip Platelet mitochondrial dysfunction of DM rats and DM patients ????Int J Clin Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=26221230 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26221230 #FOAvip English Wikipedia <ref>{{Cite pmid|26221230}}</ref> Platelet mitochondrial dysfunction of DM rats and DM patients #MMPMID26221230 Wu F; Liu Y; Luo L; Lu Y; Yew DT; Xu J; Guo K Int J Clin Exp Med 2015[]; 8 (5): 6937-46 PMID26221230show ga It is known that cardiovascular complications plays important roles in the development of diabetes mellitus (DM) and platelet dysfunction is one of the key reasons which led to microangiopathy. This study was designed to investigate the mitochondria function changes of platelet in DM rats and DM patients. The results showed that the platelets viability, platelet adenosine triphosphate (ATP) content and platelet mitochondrial ATP content of DM rats were lower than that of normal rats; when incubated in vitro for 24 h, platelet number and mitochondrial membrane potential (MMP) of DM rats were lower than that of normal rats, reactive oxygen species (ROS) was higher than that of normal rats. For DM patients, their platelet number and ROS were higher and MMP was lower than those of normal people; when incubated in vitro for 24 h, platelet viability of DM patients was lower than that of normal people. Platelet ultra-microstructures of DM rats and DM patients were abnormal. These results suggested that platelet mitochondrial function of both DM rats and DM patients was impaired when compared to normal rats and normal people, respectively. Platelets may be applied as a biomarker to observe the mitochondrial changes during DM. äPlatelet mitochondrial dysfunction of DM rats and DM patients (epmc).pdf DeepDyve Pubget Overpricing