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10.1016/j.semnephrol.2015.01.002 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2015.01.002 C4507081!4507081!25795495     free     free     free       Semin+Nephrol 2015 ; 35 (1): 2-11 Nephropedia Template TP {{tp|p=25795495|t=2015. Sepsis-Associated Acute Kidney Injury |pdf=|usr=}}{{25795495}} gab.com Text Sepsis-Associated Acute Kidney Injury JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25795495 #FOAvip Acute kidney injury (AKI) is an epidemic problem. Sepsis has long been recognized as a foremost precipitant of AKI. Sepsis-associated AKI (SA-AKI) portends a high burden of morbidity and mortality in both children and adults with critical illness. Although our understanding of its pathophysiology is incomplete, SA-AKI likely represents a distinct subset of AKI contributed to by a unique constellation of hemodynamic, inflammatory, and immune mechanisms. SA-AKI poses significant clinical challenges for clinicians. To date, no singular effective therapy has been developed to alter the natural history of SA-AKI. Rather, current strategies to alleviate poor outcomes focus on clinical risk identification, early detection of injury, modifying clinician behavior to avoid harm, early appropriate antimicrobial therapy, and surveillance among survivors for the longer-term sequelae of kidney damage. Recent evidence has confirmed that patients no longer die with AKI, but from AKI. To improve the care and outcomes for sufferers of SA-AKI, clinicians need a robust appreciation for its epidemiology and current best-evidence strategies for prevention and treatment. Twit Text FOAVip Sepsis-Associated Acute Kidney Injury ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25795495 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25795495 #FOAvip English Wikipedia <ref>{{Cite pmid|25795495}}</ref> Sepsis-Associated Acute Kidney Injury #MMPMID25795495 Alobaidi R; Basu RK; Goldstein SL; Bagshaw SM Semin Nephrol 2015[Jan]; 35 (1): 2-11 PMID25795495show ga Acute kidney injury (AKI) is an epidemic problem. Sepsis has long been recognized as a foremost precipitant of AKI. Sepsis-associated AKI (SA-AKI) portends a high burden of morbidity and mortality in both children and adults with critical illness. Although our understanding of its pathophysiology is incomplete, SA-AKI likely represents a distinct subset of AKI contributed to by a unique constellation of hemodynamic, inflammatory, and immune mechanisms. SA-AKI poses significant clinical challenges for clinicians. To date, no singular effective therapy has been developed to alter the natural history of SA-AKI. Rather, current strategies to alleviate poor outcomes focus on clinical risk identification, early detection of injury, modifying clinician behavior to avoid harm, early appropriate antimicrobial therapy, and surveillance among survivors for the longer-term sequelae of kidney damage. Recent evidence has confirmed that patients no longer die with AKI, but from AKI. To improve the care and outcomes for sufferers of SA-AKI, clinicians need a robust appreciation for its epidemiology and current best-evidence strategies for prevention and treatment. äSepsis-Associated Acute Kidney Injury (epmc).pdf DeepDyve Pubget Overpricing