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10.1007/s10863-015-9600-5 http://scihub22266oqcxt.onion/10.1007/s10863-015-9600-5 C4506732!4506732!25595872     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Bioenerg+Biomembr 2016 ; 48 (2): 169-74 Nephropedia Template TP {{tp|p=25595872|t=2016. Lipid Peroxidation in Brain or Spinal Cord Mitochondria After Injury |pdf=|usr=}}{{25595872}} gab.com Text Lipid Peroxidation in Brain or Spinal Cord Mitochondria After Injury JO: J Bioenerg Biomembr http://www.kidney.de/pget.php?&tw=1&pmid=25595872 #FOAvip Extensive evidence has demonstrated an important role of oxygen radical formation (i.e. oxidative stress) as a mediator of the secondary injury process that occurs following primary mechanical injury to the brain or spinal cord. The predominant form of oxygen radical-induced oxidative damage that occurs in injured nervous tissue is lipid peroxidation (LP). Much of the oxidative stress in injured nerve cells initially begins in mitochondria via the generation of the reactive nitrogen species peroxynitrite (PN) which then can generate multiple highly reactive free radicals including nitrogen dioxide (?NO2), hydroxyl radical (?OH) and carbonate radical (?CO3). Each can readily induce LP within the phospholipid membranes of the mitochondrion leading to respiratory dysfunction, calcium buffering impairment, mitochondrial permeability transition and cell death. Validation of the role of LP in central nervous system secondary injury has been provided by the mitochondrial and neuroprotective effects of multiple antioxidant agents which are briefly reviewed. Twit Text FOAVip Lipid Peroxidation in Brain or Spinal Cord Mitochondria After Injury ????J Bioenerg Biomembr http://www.kidney.de/pget.php?&tw=1&pmid=25595872 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25595872 #FOAvip English Wikipedia <ref>{{Cite pmid|25595872}}</ref> Lipid Peroxidation in Brain or Spinal Cord Mitochondria After Injury #MMPMID25595872 Hall ED; Wang JA; Bosken JM; Singh IN J Bioenerg Biomembr 2016[Apr]; 48 (2): 169-74 PMID25595872show ga Extensive evidence has demonstrated an important role of oxygen radical formation (i.e. oxidative stress) as a mediator of the secondary injury process that occurs following primary mechanical injury to the brain or spinal cord. The predominant form of oxygen radical-induced oxidative damage that occurs in injured nervous tissue is lipid peroxidation (LP). Much of the oxidative stress in injured nerve cells initially begins in mitochondria via the generation of the reactive nitrogen species peroxynitrite (PN) which then can generate multiple highly reactive free radicals including nitrogen dioxide (?NO2), hydroxyl radical (?OH) and carbonate radical (?CO3). Each can readily induce LP within the phospholipid membranes of the mitochondrion leading to respiratory dysfunction, calcium buffering impairment, mitochondrial permeability transition and cell death. Validation of the role of LP in central nervous system secondary injury has been provided by the mitochondrial and neuroprotective effects of multiple antioxidant agents which are briefly reviewed. äLipid Peroxidation in Brain or Spinal Cord Mitochondria After Injury (epmc).pdf DeepDyve Pubget Overpricing