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10.1089/dia.2014.0204 http://scihub22266oqcxt.onion/10.1089/dia.2014.0204 C4504429!4504429!25879401     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Diabetes+Technol+Ther 2015 ; 17 (7): 475-81 Nephropedia Template TP {{tp|p=25879401|t=2015. Network Cluster Analysis of Protein?Protein Interaction Network?Identified Biomarker for Type 2 Diabetes |pdf=|usr=}}{{25879401}} gab.com Text Network Cluster Analysis of Protein Protein Interaction Network Identified Biomarker for Type 2 Diabetes JO: Diabetes Technol Ther http://www.kidney.de/pget.php?&tw=1&pmid=25879401 #FOAvip Type 2 diabetes mellitus (T2DM) is a complex disease that is caused by an impairment in the secretion of ?-cell insulin and by a peripheral resistance to insulin. Most patients suffering from T2DM and from obesity exhibit insulin resistance in the muscles, liver, and fat, resulting in a reduced response of these tissues to insulin. In healthy individuals, pancreatic islet ?-cells secrete insulin to regulate the increase in blood glucose levels. Once these ?-cells fail to function, T2DM develops. Despite the progress achieved in this field in recent years, the genetic causes for insulin resistance and for T2DM have not yet been fully discovered. The present study aims to characterize T2DM by comparing its gene expression with that of normal controls, as well as to identify biomarkers for early T2DM. Gene expression profiles were downloaded from the Gene Expression Omnibus, and differentially expressed genes (DEGs) were identified for type 2 diabetes. Furthermore, functional analyses were conducted for the gene ontology and for the pathway enrichment. In total, 781 DEGs were identified in the T2DM samples relative to healthy controls. These genes were found to be involved in several biological processes, including cell communication, cell proliferation, cell shape, and apoptosis. We constructed a protein?protein interaction (PPI) network, and the clusters in the PPI were analyzed by using ClusterONE. Six functional genes that may play important roles in the initiation of T2DM were identified within the network. Twit Text FOAVip Network Cluster Analysis of Protein Protein Interaction Network Identified Biomarker for Type 2 Diabetes ????Diabetes Technol Ther http://www.kidney.de/pget.php?&tw=1&pmid=25879401 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25879401 #FOAvip English Wikipedia <ref>{{Cite pmid|25879401}}</ref> Network Cluster Analysis of Protein?Protein Interaction Network?Identified Biomarker for Type 2 Diabetes #MMPMID25879401 Li Z; Qiao Z; Zheng W; Ma W Diabetes Technol Ther 2015[Jul]; 17 (7): 475-81 PMID25879401show ga Type 2 diabetes mellitus (T2DM) is a complex disease that is caused by an impairment in the secretion of ?-cell insulin and by a peripheral resistance to insulin. Most patients suffering from T2DM and from obesity exhibit insulin resistance in the muscles, liver, and fat, resulting in a reduced response of these tissues to insulin. In healthy individuals, pancreatic islet ?-cells secrete insulin to regulate the increase in blood glucose levels. Once these ?-cells fail to function, T2DM develops. Despite the progress achieved in this field in recent years, the genetic causes for insulin resistance and for T2DM have not yet been fully discovered. The present study aims to characterize T2DM by comparing its gene expression with that of normal controls, as well as to identify biomarkers for early T2DM. Gene expression profiles were downloaded from the Gene Expression Omnibus, and differentially expressed genes (DEGs) were identified for type 2 diabetes. Furthermore, functional analyses were conducted for the gene ontology and for the pathway enrichment. In total, 781 DEGs were identified in the T2DM samples relative to healthy controls. These genes were found to be involved in several biological processes, including cell communication, cell proliferation, cell shape, and apoptosis. We constructed a protein?protein interaction (PPI) network, and the clusters in the PPI were analyzed by using ClusterONE. Six functional genes that may play important roles in the initiation of T2DM were identified within the network. äNetwork Cluster Analysis of Protein?Protein Interaction Network?Identi(epmc).pdf DeepDyve Pubget Overpricing