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10.1038/nrneph.2009.192 http://scihub22266oqcxt.onion/10.1038/nrneph.2009.192 C4503388!4503388!19918255     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=19918255&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Nat+Rev+Nephrol 2010 ; 6 (1): 32-40 Nephropedia Template TP {{tp|p=19918255|t=2010. Bone disease after renal transplantation |pdf=|usr=}}{{19918255}} gab.com Text Bone disease after renal transplantation JO: Nat Rev Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=19918255 #FOAvip In light of greatly improved long-term patient and graft survival after renal transplantation, improving other clinical outcomes such as risk of fracture and cardiovascular disease is of paramount importance. After renal transplantation, a large percentage of patients lose bone. This loss of bone results from a combination of factors that include pre-existing renal osteodystrophy, immunosuppressive therapy, and the effects of chronically reduced renal function after transplantation. In addition to low bone volume, histological abnormalities include decreased bone turnover and defective mineralization. Low bone volume and low bone turnover were recently shown to be associated with cardiovascular calcifications, highlighting specific challenges for medical therapy and the need to prevent low bone turnover in the pretransplant patient. This Review discusses changes in bone histology and mineral metabolism that are associated with renal transplantation and the effects of these changes on clinical outcomes such as fractures and cardiovascular calcifications. Therapeutic modalities are evaluated based on our understanding of bone histology. Twit Text FOAVip Bone disease after renal transplantation ????Nat Rev Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=19918255 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19918255 #FOAvip English Wikipedia <ref>{{Cite pmid|19918255}}</ref> Bone disease after renal transplantation #MMPMID19918255 Malluche HH; Monier-Faugere MC; Herberth J Nat Rev Nephrol 2010[Jan]; 6 (1): 32-40 PMID19918255show ga In light of greatly improved long-term patient and graft survival after renal transplantation, improving other clinical outcomes such as risk of fracture and cardiovascular disease is of paramount importance. After renal transplantation, a large percentage of patients lose bone. This loss of bone results from a combination of factors that include pre-existing renal osteodystrophy, immunosuppressive therapy, and the effects of chronically reduced renal function after transplantation. In addition to low bone volume, histological abnormalities include decreased bone turnover and defective mineralization. Low bone volume and low bone turnover were recently shown to be associated with cardiovascular calcifications, highlighting specific challenges for medical therapy and the need to prevent low bone turnover in the pretransplant patient. This Review discusses changes in bone histology and mineral metabolism that are associated with renal transplantation and the effects of these changes on clinical outcomes such as fractures and cardiovascular calcifications. Therapeutic modalities are evaluated based on our understanding of bone histology. äBone disease after renal transplantation (epmc).pdf DeepDyve Pubget Overpricing