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10.1111/j.1749-6632.2011.06320.x http://scihub22266oqcxt.onion/10.1111/j.1749-6632.2011.06320.x C4501013!4501013 !22211895     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\22211895 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Ann+N+Y+Acad+Sci 2011 ; 1243 (ä): 88-102 Nephropedia Template TP {{tp|p=22211895 |t=2011. Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications |pdf=|usr=}}{{22211895 }} gab.com Text Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications JO: Ann N Y Acad Sci http://www.kidney.de/pget.php?&tw=1&pmid=22211895 #FOAvip The receptor for advanced glycation endproducts (RAGE) was first described as a signal transduction receptor for advanced glycation endproducts (AGEs), the products of nonenzymatic glycation and oxidation of proteins and lipids that accumulate in diabetes and in inflammatory foci. The discovery that RAGE was a receptor for inflammatory S100/calgranulins and high mobility group box 1 (HMGB1) set the stage for linking RAGE to both the consequences and causes of types 1 and 2 diabetes. Recent discoveries regarding the structure of RAGE as well as novel intracellular binding partner interactions advance our understanding of the mechanisms by which RAGE evokes pathological consequences and underscore strategies by which antagonism of RAGE in the clinic may be realized. Finally, recent data tracking RAGE in the clinic suggest that levels of soluble RAGEs and polymorphisms in the gene encoding RAGE may hold promise for the identification of patients who are vulnerable to the complications of diabetes and/or are receptive to therapeutic interventions designed to prevent and reverse the damage inflicted by chronic hyperglycemia, irrespective of its etiology. Twit Text FOAVip Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications ????Ann N Y Acad Sci http://www.kidney.de/pget.php?&tw=1&pmid=22211895 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22211895 #FOAvip English Wikipedia <ref>{{Cite pmid|22211895 }}</ref> Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications #MMPMID22211895 Ramasamy R ; Yan SF ; Schmidt AM Ann N Y Acad Sci 2011[Dec]; 1243 (ä): 88-102 PMID22211895 show ga The receptor for advanced glycation endproducts (RAGE) was first described as a signal transduction receptor for advanced glycation endproducts (AGEs), the products of nonenzymatic glycation and oxidation of proteins and lipids that accumulate in diabetes and in inflammatory foci. The discovery that RAGE was a receptor for inflammatory S100/calgranulins and high mobility group box 1 (HMGB1) set the stage for linking RAGE to both the consequences and causes of types 1 and 2 diabetes. Recent discoveries regarding the structure of RAGE as well as novel intracellular binding partner interactions advance our understanding of the mechanisms by which RAGE evokes pathological consequences and underscore strategies by which antagonism of RAGE in the clinic may be realized. Finally, recent data tracking RAGE in the clinic suggest that levels of soluble RAGEs and polymorphisms in the gene encoding RAGE may hold promise for the identification of patients who are vulnerable to the complications of diabetes and/or are receptive to therapeutic interventions designed to prevent and reverse the damage inflicted by chronic hyperglycemia, irrespective of its etiology. |Animals [MESH] |Cardiovascular Diseases/etiology/metabolism [MESH] |Carrier Proteins/metabolism [MESH] |Diabetes Complications/etiology/genetics/metabolism [MESH] |Diabetes Mellitus/*etiology/genetics/*metabolism [MESH] |Diabetic Nephropathies/etiology/metabolism [MESH] |Formins [MESH] |Humans [MESH] |Inflammation/etiology/metabolism [MESH] |Mice [MESH] |Receptor for Advanced Glycation End Products [MESH] |Receptors, Immunologic/chemistry/genetics/*metabolism [MESH]Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of d(epmc).pdf DeepDyve Pubget Overpricing