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10.1146/annurev-neuro-062012-170354 http://scihub22266oqcxt.onion/10.1146/annurev-neuro-062012-170354 C4486472!4486472!25032496     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25032496&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Annu+Rev+Neurosci 2014 ; 37 (ä): 221-42 Nephropedia Template TP {{tp|p=25032496|t=2014. Neural Tube Defects |pdf=|usr=}}{{25032496}} gab.com Text Neural Tube Defects JO: Annu Rev Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25032496 #FOAvip Neural tube defects (NTDs), including spina bifida and anencephaly, are severe birth defects of the central nervous system that originate during embryonic development when the neural tube fails to close completely. Human NTDs are multifactorial, with contributions from both genetic and environmental factors. The genetic basis is not yet well understood, but several nongenetic risk factors have been identified as have possibilities for prevention by maternal folic acid supplementation. Mechanisms underlying neural tube closure and NTDs may be informed by experimental models, which have revealed numerous genes whose abnormal function causes NTDs and have provided details of critical cellular and morphological events whose regulation is essential for closure. Such models also provide an opportunity to investigate potential risk factors and to develop novel preventive therapies. Twit Text FOAVip Neural Tube Defects ????Annu Rev Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25032496 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25032496 #FOAvip English Wikipedia <ref>{{Cite pmid|25032496}}</ref> Neural Tube Defects #MMPMID25032496 Greene ND; Copp AJ Annu Rev Neurosci 2014[]; 37 (ä): 221-42 PMID25032496show ga Neural tube defects (NTDs), including spina bifida and anencephaly, are severe birth defects of the central nervous system that originate during embryonic development when the neural tube fails to close completely. Human NTDs are multifactorial, with contributions from both genetic and environmental factors. The genetic basis is not yet well understood, but several nongenetic risk factors have been identified as have possibilities for prevention by maternal folic acid supplementation. Mechanisms underlying neural tube closure and NTDs may be informed by experimental models, which have revealed numerous genes whose abnormal function causes NTDs and have provided details of critical cellular and morphological events whose regulation is essential for closure. Such models also provide an opportunity to investigate potential risk factors and to develop novel preventive therapies. äNeural Tube Defects (epmc).pdf DeepDyve Pubget Overpricing