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10.1146/annurev-immunol-031210-101352 http://scihub22266oqcxt.onion/10.1146/annurev-immunol-031210-101352 C4480235!4480235!22224766     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Annu+Rev+Immunol 2012 ; 30 (ä): 491-529 Nephropedia Template TP {{tp|p=22224766|t=2012. Signaling by myeloid C-type lectin receptors in immunity and homeostasis |pdf=|usr=}}{{22224766}} gab.com Text Signaling by myeloid C-type lectin receptors in immunity and homeostasis JO: Annu Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=22224766 #FOAvip Myeloid cells are key drivers of physiological responses to pathogen invasion or tissue damage. Members of the C-type lectin receptor (CLR) family stand out among the specialized receptors utilized by myeloid cells to orchestrate these responses. CLR ligands include carbohydrate, protein and lipid components of both pathogens and self, which variably trigger endocytic, phagocytic, pro-inflammatory or anti-inflammatory reactions. These varied outcomes rely on a versatile system for CLR signaling that includes tyrosine based motifs that recruit kinases, phosphatases or endocytic adaptors, as well as non-tyrosine based signals that modulate the activation of other pathways or couple to the uptake machinery. Here, we review the signaling properties of myeloid CLRs and how they impact the role of myeloid cells in innate and adaptive immunity. Twit Text FOAVip Signaling by myeloid C-type lectin receptors in immunity and homeostasis ????Annu Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=22224766 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22224766 #FOAvip English Wikipedia <ref>{{Cite pmid|22224766}}</ref> Signaling by myeloid C-type lectin receptors in immunity and homeostasis #MMPMID22224766 Sancho D; Reis e Sousa C Annu Rev Immunol 2012[]; 30 (ä): 491-529 PMID22224766show ga Myeloid cells are key drivers of physiological responses to pathogen invasion or tissue damage. Members of the C-type lectin receptor (CLR) family stand out among the specialized receptors utilized by myeloid cells to orchestrate these responses. CLR ligands include carbohydrate, protein and lipid components of both pathogens and self, which variably trigger endocytic, phagocytic, pro-inflammatory or anti-inflammatory reactions. These varied outcomes rely on a versatile system for CLR signaling that includes tyrosine based motifs that recruit kinases, phosphatases or endocytic adaptors, as well as non-tyrosine based signals that modulate the activation of other pathways or couple to the uptake machinery. Here, we review the signaling properties of myeloid CLRs and how they impact the role of myeloid cells in innate and adaptive immunity. äSignaling by myeloid C-type lectin receptors in immunity and homeostas(epmc).pdf DeepDyve Pubget Overpricing