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10.1016/j.gendis.2015.02.007 http://scihub22266oqcxt.onion/10.1016/j.gendis.2015.02.007 C4477834!4477834!26114157     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Genes+Dis 2015 ; 2 (2): 186-96 Nephropedia Template TP {{tp|p=26114157|t=2015. NKCC1 and NKCC2: The pathogenetic role of cation-chloride cotransporters in hypertension |pdf=|usr=}}{{26114157}} gab.com Text NKCC1 and NKCC2: The pathogenetic role of cation-chloride cotransporters in hypertension JO: Genes Dis http://www.kidney.de/pget.php?&tw=1&pmid=26114157 #FOAvip This review summarizes the data on the functional significance of ubiquitous (NKCC1) and renal-specific (NKCC2) isoforms of electroneutral sodium, potassium and chloride cotransporters. These carriers contribute to the pathogenesis of hypertension via regulation of intracellular chloride concentration in vascular smooth muscle and neuronal cells and via sensing chloride concentration in the renal tubular fluid, respectively. Both NKCC1 and NKCC2 are inhibited by furosemide and other high-ceiling diuretics widely used for attenuation of extracellular fluid volume. However, the chronic usage of these compounds for the treatment of hypertension and other volume-expanded disorders may have diverse side-effects due to suppression of myogenic response in microcirculatory beds. Twit Text FOAVip NKCC1 and NKCC2: The pathogenetic role of cation-chloride cotransporters in hypertension ????Genes Dis http://www.kidney.de/pget.php?&tw=1&pmid=26114157 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26114157 #FOAvip English Wikipedia <ref>{{Cite pmid|26114157}}</ref> NKCC1 and NKCC2: The pathogenetic role of cation-chloride cotransporters in hypertension #MMPMID26114157 Orlov SN; Koltsova SV; Kapilevich LV; Gusakova SV; Dulin NO Genes Dis 2015[Jun]; 2 (2): 186-96 PMID26114157show ga This review summarizes the data on the functional significance of ubiquitous (NKCC1) and renal-specific (NKCC2) isoforms of electroneutral sodium, potassium and chloride cotransporters. These carriers contribute to the pathogenesis of hypertension via regulation of intracellular chloride concentration in vascular smooth muscle and neuronal cells and via sensing chloride concentration in the renal tubular fluid, respectively. Both NKCC1 and NKCC2 are inhibited by furosemide and other high-ceiling diuretics widely used for attenuation of extracellular fluid volume. However, the chronic usage of these compounds for the treatment of hypertension and other volume-expanded disorders may have diverse side-effects due to suppression of myogenic response in microcirculatory beds. äNKCC1 and NKCC2: The pathogenetic role of cation-chloride cotransporte(epmc).pdf DeepDyve Pubget Overpricing