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10.1016/j.redox.2015.05.008 http://scihub22266oqcxt.onion/10.1016/j.redox.2015.05.008 C4475862!4475862 !26079210     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26079210 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Redox+Biol 2015 ; 5 (ä): 275-289 Nephropedia Template TP {{tp|p=26079210 |t=2015. Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair |pdf=|usr=}}{{26079210 }} gab.com Text Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair JO: Redox Biol http://www.kidney.de/pget.php?&tw=1&pmid=26079210 #FOAvip Reactive oxygen and nitrogen species (e.g. H2O2, nitric oxide) confer redox regulation of essential cellular signaling pathways such as cell differentiation, proliferation, migration and apoptosis. In addition, classical regulation of gene expression or activity, including gene transcription to RNA followed by translation to the protein level, by transcription factors (e.g. NF-?B, HIF-1?) and mRNA binding proteins (e.g. GAPDH, HuR) is subject to redox regulation. This review will give an update of recent discoveries in this field, and specifically highlight the impact of reactive oxygen and nitrogen species on DNA repair systems that contribute to genomic stability. Emphasis will be placed on the emerging role of redox mechanisms regulating epigenetic pathways (e.g. miRNA, DNA methylation and histone modifications). By providing clinical correlations we discuss how oxidative stress can impact on gene regulation/activity and vise versa, how epigenetic processes, other gene regulatory mechanisms and DNA repair can influence the cellular redox state and contribute or prevent development or progression of disease. Twit Text FOAVip Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair ????Redox Biol http://www.kidney.de/pget.php?&tw=1&pmid=26079210 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26079210 #FOAvip English Wikipedia <ref>{{Cite pmid|26079210 }}</ref> Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair #MMPMID26079210 Mikhed Y ; Görlach A ; Knaus UG ; Daiber A Redox Biol 2015[Aug]; 5 (ä): 275-289 PMID26079210 show ga Reactive oxygen and nitrogen species (e.g. H2O2, nitric oxide) confer redox regulation of essential cellular signaling pathways such as cell differentiation, proliferation, migration and apoptosis. In addition, classical regulation of gene expression or activity, including gene transcription to RNA followed by translation to the protein level, by transcription factors (e.g. NF-?B, HIF-1?) and mRNA binding proteins (e.g. GAPDH, HuR) is subject to redox regulation. This review will give an update of recent discoveries in this field, and specifically highlight the impact of reactive oxygen and nitrogen species on DNA repair systems that contribute to genomic stability. Emphasis will be placed on the emerging role of redox mechanisms regulating epigenetic pathways (e.g. miRNA, DNA methylation and histone modifications). By providing clinical correlations we discuss how oxidative stress can impact on gene regulation/activity and vise versa, how epigenetic processes, other gene regulatory mechanisms and DNA repair can influence the cellular redox state and contribute or prevent development or progression of disease. |*DNA Damage [MESH] |*DNA Repair [MESH] |*Epigenesis, Genetic [MESH] |*Genomic Instability [MESH] |Biomarkers/metabolism [MESH] |Gene Expression Regulation [MESH] |Histones/metabolism [MESH] |Humans [MESH] |Oxidation-Reduction [MESH] |Oxidative Stress [MESH] |RNA, Untranslated/chemistry/metabolism [MESH]Redox regulation of genome stability by effects on gene expression, ep(epmc).pdf DeepDyve Pubget Overpricing