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10.3389/fimmu.2015.00316 http://scihub22266oqcxt.onion/10.3389/fimmu.2015.00316 C4470258!4470258!26136748     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2015 ; 6 (ä): ä Nephropedia Template TP {{tp|p=26136748|t=2015. Type II NKT Cells in Inflammation, Autoimmunity, Microbial Immunity, and Cancer |pdf=|usr=}}{{26136748}} gab.com Text Type II NKT Cells in Inflammation, Autoimmunity, Microbial Immunity, and Cancer JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26136748 #FOAvip Natural killer T cells (NKT) recognize self and microbial lipid antigens presented by non-polymorphic CD1d molecules. Two major NKT cell subsets, type I and II, express different types of antigen receptors (TCR) with distinct mode of CD1d/lipid recognition. Though type II NKT cells are less frequent in mice and difficult to study, they are predominant in human. One of the major subsets of type II NKT cells reactive to the self-glycolipid sulfatide is the best characterized and has been shown to induce a dominant immune regulatory mechanism that controls inflammation in autoimmunity and in anti-cancer immunity. Recently, type II NKT cells reactive to other self-glycolipids and phospholipids have been identified suggesting both promiscuous and specific TCR recognition in microbial immunity as well. Since the CD1d pathway is highly conserved, a detailed understanding of the biology and function of type II NKT cells as well as their interplay with type I NKT cells or other innate and adaptive T cells will have major implications for potential novel interventions in inflammatory and autoimmune diseases, microbial immunity, and cancer. Twit Text FOAVip Type II NKT Cells in Inflammation, Autoimmunity, Microbial Immunity, and Cancer ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26136748 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26136748 #FOAvip English Wikipedia <ref>{{Cite pmid|26136748}}</ref> Type II NKT Cells in Inflammation, Autoimmunity, Microbial Immunity, and Cancer #MMPMID26136748 Marrero I; Ware R; Kumar V Front Immunol 2015[]; 6 (ä): ä PMID26136748show ga Natural killer T cells (NKT) recognize self and microbial lipid antigens presented by non-polymorphic CD1d molecules. Two major NKT cell subsets, type I and II, express different types of antigen receptors (TCR) with distinct mode of CD1d/lipid recognition. Though type II NKT cells are less frequent in mice and difficult to study, they are predominant in human. One of the major subsets of type II NKT cells reactive to the self-glycolipid sulfatide is the best characterized and has been shown to induce a dominant immune regulatory mechanism that controls inflammation in autoimmunity and in anti-cancer immunity. Recently, type II NKT cells reactive to other self-glycolipids and phospholipids have been identified suggesting both promiscuous and specific TCR recognition in microbial immunity as well. Since the CD1d pathway is highly conserved, a detailed understanding of the biology and function of type II NKT cells as well as their interplay with type I NKT cells or other innate and adaptive T cells will have major implications for potential novel interventions in inflammatory and autoimmune diseases, microbial immunity, and cancer. äType II NKT Cells in Inflammation, Autoimmunity, Microbial Immunity, a(epmc).pdf DeepDyve Pubget Overpricing