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10.1186/s40364-015-0034-3 http://scihub22266oqcxt.onion/10.1186/s40364-015-0034-3 C4469398!4469398!26085929     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 251.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomark+Res 2015 ; 3 (ä): ä Nephropedia Template TP {{tp|p=26085929|t=2015. Glioblastoma vasculogenic mimicry: signaling pathways progression and potential anti-angiogenesis targets |pdf=|usr=}}{{26085929}} gab.com Text Glioblastoma vasculogenic mimicry: signaling pathways progression and potential anti-angiogenesis targets JO: Biomark Res http://www.kidney.de/pget.php?&tw=1&pmid=26085929 #FOAvip Glioblastoma (GBM) is a highly angiogenic malignancy that is resistant to standard therapy; neo-formed vessels of this aggressive malignancy are thought to arise by sprouting of pre-existing brain capillaries. However, the conventional anti-angiogenic therapy, which seemed promising initially, shows transitory and incomplete efficacy. The discovery of vasculogenic mimicry (VM) has offered a new horizon for understanding tumor vascularization. VM is a tumor cell-constituted, matrix-embedded fluid-conducting meshwork that is independent of endothelial cells and is positively correlated with poor prognosis. Therefore, a better understanding of GBM vasculature is needed to optimize anti-angiogenic therapy. This review focuses on the signaling molecules and cascades involved in VM in relation to ongoing glioma research, as well as the clinical translational advances in GBM that have been offered by the development of optimized anti-angiogenesis treatment modalities. Twit Text FOAVip Glioblastoma vasculogenic mimicry: signaling pathways progression and potential anti-angiogenesis targets ????Biomark Res http://www.kidney.de/pget.php?&tw=1&pmid=26085929 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26085929 #FOAvip English Wikipedia <ref>{{Cite pmid|26085929}}</ref> Glioblastoma vasculogenic mimicry: signaling pathways progression and potential anti-angiogenesis targets #MMPMID26085929 Mao Jm; Liu J; Guo G; Mao Xg; Li Cx Biomark Res 2015[]; 3 (ä): ä PMID26085929show ga Glioblastoma (GBM) is a highly angiogenic malignancy that is resistant to standard therapy; neo-formed vessels of this aggressive malignancy are thought to arise by sprouting of pre-existing brain capillaries. However, the conventional anti-angiogenic therapy, which seemed promising initially, shows transitory and incomplete efficacy. The discovery of vasculogenic mimicry (VM) has offered a new horizon for understanding tumor vascularization. VM is a tumor cell-constituted, matrix-embedded fluid-conducting meshwork that is independent of endothelial cells and is positively correlated with poor prognosis. Therefore, a better understanding of GBM vasculature is needed to optimize anti-angiogenic therapy. This review focuses on the signaling molecules and cascades involved in VM in relation to ongoing glioma research, as well as the clinical translational advances in GBM that have been offered by the development of optimized anti-angiogenesis treatment modalities. äGlioblastoma vasculogenic mimicry: signaling pathways progression and (epmc).pdf DeepDyve Pubget Overpricing