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10.1155/2015/508409 http://scihub22266oqcxt.onion/10.1155/2015/508409 C4468292!4468292!26136779     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Endocrinol 2015 ; 2015 (ä): ä Nephropedia Template TP {{tp|p=26136779|t=2015. Mechanisms Linking Inflammation to Insulin Resistance |pdf=|usr=}}{{26136779}} gab.com Text Mechanisms Linking Inflammation to Insulin Resistance JO: Int J Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26136779 #FOAvip Obesity is now widespread around the world. Obesity-associated chronic low-grade inflammation is responsible for the decrease of insulin sensitivity, which makes obesity a major risk factor for insulin resistance and related diseases such as type 2 diabetes mellitus and metabolic syndromes. The state of low-grade inflammation is caused by overnutrition which leads to lipid accumulation in adipocytes. Obesity might increase the expression of some inflammatory cytokines and activate several signaling pathways, both of which are involved in the pathogenesis of insulin resistance by interfering with insulin signaling and action. It has been suggested that specific factors and signaling pathways are often correlated with each other; therefore, both of the fluctuation of cytokines and the status of relevant signaling pathways should be considered during studies analyzing inflammation-related insulin resistance. In this paper, we discuss how these factors and signaling pathways contribute to insulin resistance and the therapeutic promise targeting inflammation in insulin resistance based on the latest experimental studies. Twit Text FOAVip Mechanisms Linking Inflammation to Insulin Resistance ????Int J Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26136779 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26136779 #FOAvip English Wikipedia <ref>{{Cite pmid|26136779}}</ref> Mechanisms Linking Inflammation to Insulin Resistance #MMPMID26136779 Chen L; Chen R; Wang H; Liang F Int J Endocrinol 2015[]; 2015 (ä): ä PMID26136779show ga Obesity is now widespread around the world. Obesity-associated chronic low-grade inflammation is responsible for the decrease of insulin sensitivity, which makes obesity a major risk factor for insulin resistance and related diseases such as type 2 diabetes mellitus and metabolic syndromes. The state of low-grade inflammation is caused by overnutrition which leads to lipid accumulation in adipocytes. Obesity might increase the expression of some inflammatory cytokines and activate several signaling pathways, both of which are involved in the pathogenesis of insulin resistance by interfering with insulin signaling and action. It has been suggested that specific factors and signaling pathways are often correlated with each other; therefore, both of the fluctuation of cytokines and the status of relevant signaling pathways should be considered during studies analyzing inflammation-related insulin resistance. In this paper, we discuss how these factors and signaling pathways contribute to insulin resistance and the therapeutic promise targeting inflammation in insulin resistance based on the latest experimental studies. äMechanisms Linking Inflammation to Insulin Resistance (epmc).pdf DeepDyve Pubget Overpricing