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10.1073/pnas.1424236112 http://scihub22266oqcxt.onion/10.1073/pnas.1424236112 C4466705!4466705!25941360     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+Natl+Acad+Sci+U+S+A 2015 ; 112 (23): 7249-54 Nephropedia Template TP {{tp|p=25941360|t=2015. Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33 |pdf=|usr=}}{{25941360}} gab.com Text Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33 JO: Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=25941360 #FOAvip The association of hypertension and inflammation is clear, but mechanisms for this human finding remain elusive. The experiments in this article demonstrate that a highly local intramyocardial signaling pathway through ST2, the receptor for the proinflammatory cytokine interleukin 33 (IL-33), regulates the heart?s response to pressure overload. By generating and using new conditional deletion mice, we identified endothelial cells as the major source of systemic circulating IL-33. Our study reveals that endothelial cell secretion of IL-33 is crucial for translating myocardial pressure overload into a selective systemic inflammatory state. Twit Text FOAVip Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33 ????Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=25941360 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25941360 #FOAvip English Wikipedia <ref>{{Cite pmid|25941360}}</ref> Myocardial pressure overload induces systemic inflammation through endothelial cell IL-33 #MMPMID25941360 Chen WY; Hong J; Gannon J; Kakkar R; Lee RT Proc Natl Acad Sci U S A 2015[Jun]; 112 (23): 7249-54 PMID25941360show ga The association of hypertension and inflammation is clear, but mechanisms for this human finding remain elusive. The experiments in this article demonstrate that a highly local intramyocardial signaling pathway through ST2, the receptor for the proinflammatory cytokine interleukin 33 (IL-33), regulates the heart?s response to pressure overload. By generating and using new conditional deletion mice, we identified endothelial cells as the major source of systemic circulating IL-33. Our study reveals that endothelial cell secretion of IL-33 is crucial for translating myocardial pressure overload into a selective systemic inflammatory state. äMyocardial pressure overload induces systemic inflammation through end(epmc).pdf DeepDyve Pubget Overpricing