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10.1002/jbmr.2373 http://scihub22266oqcxt.onion/10.1002/jbmr.2373 C4455538!4455538 !25264148     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25264148 &cmd=llinks): Failed to open stream: HTTP request failed! 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The convergence of fracture repair and stem cells: interplay of genes, aging, environmental factors and disease |pdf=|usr=}}{{25264148 }} gab.com Text The convergence of fracture repair and stem cells: interplay of genes, aging, environmental factors and disease JO: J Bone Miner Res http://www.kidney.de/pget.php?&tw=1&pmid=25264148 #FOAvip The complexity of fracture repair makes it an ideal process for studying the interplay between the molecular, cellular, tissue, and organ level events involved in tissue regeneration. Additionally, as fracture repair recapitulates many of the processes that occur during embryonic development, investigations of fracture repair provide insights regarding skeletal embryogenesis. Specifically, inflammation, signaling, gene expression, cellular proliferation and differentiation, osteogenesis, chondrogenesis, angiogenesis, and remodeling represent the complex array of interdependent biological events that occur during fracture repair. Here we review studies of bone regeneration in genetically modified mouse models, during aging, following environmental exposure, and in the setting of disease that provide insights regarding the role of multipotent cells and their regulation during fracture repair. Complementary animal models and ongoing scientific discoveries define an increasing number of molecular and cellular targets to reduce the morbidity and complications associated with fracture repair. Last, some new and exciting areas of stem cell research such as the contribution of mitochondria function, limb regeneration signaling, and microRNA (miRNA) posttranscriptional regulation are all likely to further contribute to our understanding of fracture repair as an active branch of regenerative medicine. Twit Text FOAVip The convergence of fracture repair and stem cells: interplay of genes, aging, environmental factors and disease ????J Bone Miner Res http://www.kidney.de/pget.php?&tw=1&pmid=25264148 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25264148 #FOAvip English Wikipedia <ref>{{Cite pmid|25264148 }}</ref> The convergence of fracture repair and stem cells: interplay of genes, aging, environmental factors and disease #MMPMID25264148 Hadjiargyrou M ; O'Keefe RJ J Bone Miner Res 2014[Nov]; 29 (11 ): 2307-22 PMID25264148 show ga The complexity of fracture repair makes it an ideal process for studying the interplay between the molecular, cellular, tissue, and organ level events involved in tissue regeneration. Additionally, as fracture repair recapitulates many of the processes that occur during embryonic development, investigations of fracture repair provide insights regarding skeletal embryogenesis. Specifically, inflammation, signaling, gene expression, cellular proliferation and differentiation, osteogenesis, chondrogenesis, angiogenesis, and remodeling represent the complex array of interdependent biological events that occur during fracture repair. Here we review studies of bone regeneration in genetically modified mouse models, during aging, following environmental exposure, and in the setting of disease that provide insights regarding the role of multipotent cells and their regulation during fracture repair. Complementary animal models and ongoing scientific discoveries define an increasing number of molecular and cellular targets to reduce the morbidity and complications associated with fracture repair. Last, some new and exciting areas of stem cell research such as the contribution of mitochondria function, limb regeneration signaling, and microRNA (miRNA) posttranscriptional regulation are all likely to further contribute to our understanding of fracture repair as an active branch of regenerative medicine. |*Bone Regeneration [MESH] |*Gene Expression Regulation [MESH] |*Signal Transduction [MESH] |Aging/genetics/*metabolism/pathology [MESH] |Animals [MESH] |Fractures, Bone/genetics/*metabolism/pathology [MESH] |Humans [MESH] |Mice [MESH] |Mice, Transgenic [MESH] |MicroRNAs/biosynthesis/genetics [MESH]The convergence of fracture repair and stem cells: interplay of genes,(epmc).pdf DeepDyve Pubget Overpricing