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10.1016/j.taap.2013.12.022 http://scihub22266oqcxt.onion/10.1016/j.taap.2013.12.022 C4455034!4455034!24398106     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24398106&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Toxicol+Appl+Pharmacol 2014 ; 275 (2): 145-51 Nephropedia Template TP {{tp|p=24398106|t=2014. Oxidative stress-induced autophagy: Role in pulmonary toxicity |pdf=|usr=}}{{24398106}} gab.com Text Oxidative stress-induced autophagy: Role in pulmonary toxicity JO: Toxicol Appl Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=24398106 #FOAvip Autophagy is an evolutionarily conserved catabolic process important in regulating the turnover of essential proteins and in elimination of damaged organelles and protein aggregates. Autophagy is observed in the lung in response to oxidative stress generated as a consequence of exposure to environmental toxicants. Whether autophagy plays role in promoting cell survival or cytotoxicity is unclear. In this article recent findings on oxidative stress-induced autophagy in the lung are reviewed; potential mechanisms initiating autophagy are also discussed. A better understanding of autophagy and its role in pulmonary toxicity may lead to the development of new strategies to treat lung injury associated with oxidative stress. Twit Text FOAVip Oxidative stress-induced autophagy: Role in pulmonary toxicity ????Toxicol Appl Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=24398106 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24398106 #FOAvip English Wikipedia <ref>{{Cite pmid|24398106}}</ref> Oxidative stress-induced autophagy: Role in pulmonary toxicity #MMPMID24398106 Malaviya R; Laskin JD; Laskin DL Toxicol Appl Pharmacol 2014[Mar]; 275 (2): 145-51 PMID24398106show ga Autophagy is an evolutionarily conserved catabolic process important in regulating the turnover of essential proteins and in elimination of damaged organelles and protein aggregates. Autophagy is observed in the lung in response to oxidative stress generated as a consequence of exposure to environmental toxicants. Whether autophagy plays role in promoting cell survival or cytotoxicity is unclear. In this article recent findings on oxidative stress-induced autophagy in the lung are reviewed; potential mechanisms initiating autophagy are also discussed. A better understanding of autophagy and its role in pulmonary toxicity may lead to the development of new strategies to treat lung injury associated with oxidative stress. äOxidative stress-induced autophagy: Role in pulmonary toxicity (epmc).pdf DeepDyve Pubget Overpricing