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10.1091/mbc.E14-09-1413

http://scihub22266oqcxt.onion/10.1091/mbc.E14-09-1413
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C4454184!4454184!25631822
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suck abstract from ncbi


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pmid25631822      Mol+Biol+Cell 2015 ; 26 (7): 1395-410
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  • Heterochromatin formation via recruitment of DNA repair proteins #MMPMID25631822
  • Kirkland JG; Peterson MR; Still CD; Brueggeman L; Dhillon N; Kamakaka RT
  • Mol Biol Cell 2015[Apr]; 26 (7): 1395-410 PMID25631822show ga
  • Double-strand-break repair proteins interact with and recruit Sir proteins to ectopic sites in the genome. Recruitment results in gene silencing, which depends on Sir proteins, as well as on histone H2A modification. Silencing also results in the localization of the locus to the nuclear periphery.
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