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10.1016/j.semnephrol.2014.08.007 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2014.08.007 C4450367!4450367 !25416662     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25416662 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Semin+Nephrol 2014 ; 34 (5 ): 532-9 Nephropedia Template TP {{tp|p=25416662 |t=2014. Pathophysiology and treatment of resistant hypertension: the role of aldosterone and amiloride-sensitive sodium channels |pdf=|usr=}}{{25416662 }} gab.com Text Pathophysiology and treatment of resistant hypertension: the role of aldosterone and amiloride-sensitive sodium channels JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25416662 #FOAvip Resistant hypertension is a clinically distinct subgroup of hypertension defined by the failure to achieve blood pressure control on optimal dosing of at least 3 antihypertensive medications of different classes, including a diuretic. The pathophysiology of hypertension can be attributed to aldosterone excess in more than 20% of patients with resistant hypertension. Existing dogma attributes the increase in blood pressure seen with increases in aldosterone to its antinatriuretic effects in the distal nephron. However, emerging research, which has identified and has begun to define the function of amiloride-sensitive sodium channels and mineralocorticoid receptors in the systemic vasculature, challenges impaired natriuresis as the sole cause of aldosterone-mediated resistant hypertension. This review integrates these findings to better define the role of the vasculature and aldosterone in the pathophysiology of resistant hypertension. In addition, a brief guide to the treatment of resistant hypertension is presented. Twit Text FOAVip Pathophysiology and treatment of resistant hypertension: the role of aldosterone and amiloride-sensitive sodium channels ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25416662 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25416662 #FOAvip English Wikipedia <ref>{{Cite pmid|25416662 }}</ref> Pathophysiology and treatment of resistant hypertension: the role of aldosterone and amiloride-sensitive sodium channels #MMPMID25416662 Judd EK ; Calhoun DA ; Warnock DG Semin Nephrol 2014[]; 34 (5 ): 532-9 PMID25416662 show ga Resistant hypertension is a clinically distinct subgroup of hypertension defined by the failure to achieve blood pressure control on optimal dosing of at least 3 antihypertensive medications of different classes, including a diuretic. The pathophysiology of hypertension can be attributed to aldosterone excess in more than 20% of patients with resistant hypertension. Existing dogma attributes the increase in blood pressure seen with increases in aldosterone to its antinatriuretic effects in the distal nephron. However, emerging research, which has identified and has begun to define the function of amiloride-sensitive sodium channels and mineralocorticoid receptors in the systemic vasculature, challenges impaired natriuresis as the sole cause of aldosterone-mediated resistant hypertension. This review integrates these findings to better define the role of the vasculature and aldosterone in the pathophysiology of resistant hypertension. In addition, a brief guide to the treatment of resistant hypertension is presented. |Aldosterone/*metabolism [MESH] |Amiloride/*pharmacology [MESH] |Antihypertensive Agents/therapeutic use [MESH] |Blood Pressure [MESH] |Coronary Vasospasm/drug therapy/metabolism [MESH] |Diuretics/*pharmacology/therapeutic use [MESH] |Drug Resistance [MESH] |Humans [MESH] |Hypertension/*drug therapy/*metabolism [MESH] |Potassium, Dietary [MESH] |Receptors, Mineralocorticoid [MESH] |Sodium Channels/*drug effects [MESH]Pathophysiology and treatment of resistant hypertension: the role of a(epmc).pdf DeepDyve Pubget Overpricing