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10.1002/iub.1383 http://scihub22266oqcxt.onion/10.1002/iub.1383 C4449288!4449288!25988524     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       IUBMB+Life 2015 ; 67 (5): 331-7 Nephropedia Template TP {{tp|p=25988524|t=2015. RSK3 ? A Regulator of Pathological Cardiac Remodeling |pdf=|usr=}}{{25988524}} gab.com Text RSK3 A Regulator of Pathological Cardiac Remodeling JO: IUBMB Life http://www.kidney.de/pget.php?&tw=1&pmid=25988524 #FOAvip The family of p90 ribosomal S6 kinases (RSK) are pleiotropic effectors for extracellular signal-regulated kinase (ERK) signaling pathways. Recently, RSK3 was shown to be important for pathological remodeling of the heart. While cardiac myocyte hypertrophy can be compensatory for increased wall stress, in chronic heart diseases this non-mitotic cell growth is usually associated with interstitial fibrosis, increased cell death, and decreased cardiac function. Although RSK3 is less abundant in the cardiac myocyte than other RSK family members, RSK3 appears to serve a unique role in cardiac myocyte stress responses. A potential mechanism conferring RSK3?s unique function in the heart is anchoring by the scaffold protein muscle A-kinase Anchoring Protein ? (mAKAP?). Recent findings suggest that RSK3 should be considered as a therapeutic target for the prevention of heart failure, a clinical syndrome of major public health significance. Twit Text FOAVip RSK3 A Regulator of Pathological Cardiac Remodeling ????IUBMB Life http://www.kidney.de/pget.php?&tw=1&pmid=25988524 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25988524 #FOAvip English Wikipedia <ref>{{Cite pmid|25988524}}</ref> RSK3 ? A Regulator of Pathological Cardiac Remodeling #MMPMID25988524 Martinez EC; Passariello CL; Li J; Matheson CJ; Dodge-Kafka K; Reigan P; Kapiloff MS IUBMB Life 2015[May]; 67 (5): 331-7 PMID25988524show ga The family of p90 ribosomal S6 kinases (RSK) are pleiotropic effectors for extracellular signal-regulated kinase (ERK) signaling pathways. Recently, RSK3 was shown to be important for pathological remodeling of the heart. While cardiac myocyte hypertrophy can be compensatory for increased wall stress, in chronic heart diseases this non-mitotic cell growth is usually associated with interstitial fibrosis, increased cell death, and decreased cardiac function. Although RSK3 is less abundant in the cardiac myocyte than other RSK family members, RSK3 appears to serve a unique role in cardiac myocyte stress responses. A potential mechanism conferring RSK3?s unique function in the heart is anchoring by the scaffold protein muscle A-kinase Anchoring Protein ? (mAKAP?). Recent findings suggest that RSK3 should be considered as a therapeutic target for the prevention of heart failure, a clinical syndrome of major public health significance. äRSK3 ? A Regulator of Pathological Cardiac Remodeling (epmc).pdf DeepDyve Pubget Overpricing