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10.1016/j.jceh.2014.02.004 http://scihub22266oqcxt.onion/10.1016/j.jceh.2014.02.004 C4442857!4442857!26041966     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Exp+Hepatol 2015 ; 5 (Suppl 1): S96-S103 Nephropedia Template TP {{tp|p=26041966|t=2015. Pathogenesis of Hepatic Encephalopathy and Brain Edema in Acute Liver Failure |pdf=|usr=}}{{26041966}} gab.com Text Pathogenesis of Hepatic Encephalopathy and Brain Edema in Acute Liver Failure JO: J Clin Exp Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=26041966 #FOAvip Neuropathologic investigations in acute liver failure (ALF) reveal significant alterations to neuroglia consisting of swelling of astrocytes leading to cytotoxic brain edema and intracranial hypertension as well as activation of microglia indicative of a central neuroinflammatory response. Increased arterial ammonia concentrations in patients with ALF are predictors of patients at risk for the development of brain herniation. Molecular and spectroscopic techniques in ALF reveal alterations in expression of an array of genes coding for neuroglial proteins involved in cell volume regulation and mitochondrial function as well as in the transport of neurotransmitter amino acids and in the synthesis of pro-inflammatory cytokines. Liver-brain pro-inflammatory signaling mechanisms involving transduction of systemically-derived cytokines, ammonia neurotoxicity and exposure to increased brain lactate have been proposed. Mild hypothermia and N-Acetyl cysteine have both hepato-protective and neuro-protective properties in ALF. Potentially effective anti-inflammatory agents aimed at control of encephalopathy and brain edema in ALF include etanercept and the antibiotic minocycline, a potent inhibitor of microglial activation. Translation of these potentially-interesting findings to the clinic is anxiously awaited. Twit Text FOAVip Pathogenesis of Hepatic Encephalopathy and Brain Edema in Acute Liver Failure ????J Clin Exp Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=26041966 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26041966 #FOAvip English Wikipedia <ref>{{Cite pmid|26041966}}</ref> Pathogenesis of Hepatic Encephalopathy and Brain Edema in Acute Liver Failure #MMPMID26041966 Butterworth RF J Clin Exp Hepatol 2015[Mar]; 5 (Suppl 1): S96-S103 PMID26041966show ga Neuropathologic investigations in acute liver failure (ALF) reveal significant alterations to neuroglia consisting of swelling of astrocytes leading to cytotoxic brain edema and intracranial hypertension as well as activation of microglia indicative of a central neuroinflammatory response. Increased arterial ammonia concentrations in patients with ALF are predictors of patients at risk for the development of brain herniation. Molecular and spectroscopic techniques in ALF reveal alterations in expression of an array of genes coding for neuroglial proteins involved in cell volume regulation and mitochondrial function as well as in the transport of neurotransmitter amino acids and in the synthesis of pro-inflammatory cytokines. Liver-brain pro-inflammatory signaling mechanisms involving transduction of systemically-derived cytokines, ammonia neurotoxicity and exposure to increased brain lactate have been proposed. Mild hypothermia and N-Acetyl cysteine have both hepato-protective and neuro-protective properties in ALF. Potentially effective anti-inflammatory agents aimed at control of encephalopathy and brain edema in ALF include etanercept and the antibiotic minocycline, a potent inhibitor of microglial activation. Translation of these potentially-interesting findings to the clinic is anxiously awaited. äPathogenesis of Hepatic Encephalopathy and Brain Edema in Acute Liver (epmc).pdf DeepDyve Pubget Overpricing