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10.1093/ckj/sfv020 http://scihub22266oqcxt.onion/10.1093/ckj/sfv020 C4440467!4440467!26034600     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Kidney+J 2015 ; 8 (3): 343-50 Nephropedia Template TP {{tp|p=26034600|t=2015. The kidneys and ANCA-associated vasculitis: from pathogenesis to diagnosis |pdf=|usr=}}{{26034600}} gab.com Text The kidneys and ANCA-associated vasculitis: from pathogenesis to diagnosis JO: Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26034600 #FOAvip Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides (AAV) are a group of pauci-immune small vessel vasculitides that often affect the kidneys manifesting as rapidly progressive glomerulonephritis. Although the exact pathogenesis of AAV is not fully known, evidence from in vitro, in vivo and clinical studies all point to the involvement of ANCA in the pathogenesis of AAV. In this review, we highlight the contributory roles played by various factors (e.g. genetics, environment, B and T-regulatory cells, toll-like receptors, etc.) in the pathogenesis of AAV. Furthermore, we discuss renal involvement in AAV in terms of clinical features and the various histopathological classification patterns, which are also known to be of prognostic importance. We also present information on useful imaging techniques for localizing kidney and other organ system involvement in AAV, and also on novel laboratory methods and assays useful for rapid and more specific determination of patients' ANCA status. Finally, we demonstrate evidence on novel serum biomarkers that have been shown to correlate with disease activity in AAV. Twit Text FOAVip The kidneys and ANCA-associated vasculitis: from pathogenesis to diagnosis ????Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26034600 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26034600 #FOAvip English Wikipedia <ref>{{Cite pmid|26034600}}</ref> The kidneys and ANCA-associated vasculitis: from pathogenesis to diagnosis #MMPMID26034600 Rowaiye OO; Kusztal M; Klinger M Clin Kidney J 2015[Jun]; 8 (3): 343-50 PMID26034600show ga Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides (AAV) are a group of pauci-immune small vessel vasculitides that often affect the kidneys manifesting as rapidly progressive glomerulonephritis. Although the exact pathogenesis of AAV is not fully known, evidence from in vitro, in vivo and clinical studies all point to the involvement of ANCA in the pathogenesis of AAV. In this review, we highlight the contributory roles played by various factors (e.g. genetics, environment, B and T-regulatory cells, toll-like receptors, etc.) in the pathogenesis of AAV. Furthermore, we discuss renal involvement in AAV in terms of clinical features and the various histopathological classification patterns, which are also known to be of prognostic importance. We also present information on useful imaging techniques for localizing kidney and other organ system involvement in AAV, and also on novel laboratory methods and assays useful for rapid and more specific determination of patients' ANCA status. Finally, we demonstrate evidence on novel serum biomarkers that have been shown to correlate with disease activity in AAV. äThe kidneys and ANCA-associated vasculitis: from pathogenesis to diagn(epmc).pdf DeepDyve Pubget Overpricing