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http://scihub22266oqcxt.onion/ C4440128!4440128!26045819     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Clin+Exp+Pathol 2015 ; 8 (3): 3069-75 Nephropedia Template TP {{tp|p=26045819|t=2015. CD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of dysferlinopathy |pdf=|usr=}}{{26045819}} gab.com Text CD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of dysferlinopathy JO: Int J Clin Exp Pathol http://www.kidney.de/pget.php?&tw=1&pmid=26045819 #FOAvip Objective: Dysferlin is a sarcolemmal protein that plays an important role in membrane repair by regulating vesicle fusion with the sarcolemma. Mutations in the dysferlin gene (DYSF) lead to multiple clinical phenotypes, including Miyoshi myopathy (MM), limb girdle muscular dystrophy type 2B (LGMD 2B), and distal myopathy with anterior tibial onset (DMAT). Patients with dysferlinopathy also show muscle inflammation, which often leads to a misdiagnosis as inflammatory myopathy. In this study, we examined and analyzed the dyferlinopathy-associated immunological features. Methods: Comparative immunohistochemical analysis of inflammatory cell infiltration, and muscle expression of MHC-I and C5b-9 was performed using muscle biopsy samples from 14 patients with dysferlinopathy, 7 patients with polymyositis, and 8 patients with either Duchenne muscular dystrophy or Becker muscular dystrophy (DMD/BMD). Results: Immunohistochemical analysis revealed positive staining for immune response-related CD4+ cells, macrophages, MHC-I and C5b-9 in dysferlinopathy, which is in a different mode of polymyositis and DMD/BMD. Conclusion: These results demonstrated the involvement of immune factors in the pathogenesis of dysferlinopathy. Twit Text FOAVip CD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of dysferlinopathy ????Int J Clin Exp Pathol http://www.kidney.de/pget.php?&tw=1&pmid=26045819 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26045819 #FOAvip English Wikipedia <ref>{{Cite pmid|26045819}}</ref> CD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of dysferlinopathy #MMPMID26045819 Yin X; Wang Q; Chen T; Niu J; Ban R; Liu J; Mao Y; Pu C Int J Clin Exp Pathol 2015[]; 8 (3): 3069-75 PMID26045819show ga Objective: Dysferlin is a sarcolemmal protein that plays an important role in membrane repair by regulating vesicle fusion with the sarcolemma. Mutations in the dysferlin gene (DYSF) lead to multiple clinical phenotypes, including Miyoshi myopathy (MM), limb girdle muscular dystrophy type 2B (LGMD 2B), and distal myopathy with anterior tibial onset (DMAT). Patients with dysferlinopathy also show muscle inflammation, which often leads to a misdiagnosis as inflammatory myopathy. In this study, we examined and analyzed the dyferlinopathy-associated immunological features. Methods: Comparative immunohistochemical analysis of inflammatory cell infiltration, and muscle expression of MHC-I and C5b-9 was performed using muscle biopsy samples from 14 patients with dysferlinopathy, 7 patients with polymyositis, and 8 patients with either Duchenne muscular dystrophy or Becker muscular dystrophy (DMD/BMD). Results: Immunohistochemical analysis revealed positive staining for immune response-related CD4+ cells, macrophages, MHC-I and C5b-9 in dysferlinopathy, which is in a different mode of polymyositis and DMD/BMD. Conclusion: These results demonstrated the involvement of immune factors in the pathogenesis of dysferlinopathy. äCD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of d(epmc).pdf DeepDyve Pubget Overpricing