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10.1093/ckj/sft031 http://scihub22266oqcxt.onion/10.1093/ckj/sft031 C4438388!4438388!26064509     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Kidney+J 2013 ; 6 (5): 457-63 Nephropedia Template TP {{tp|p=26064509|t=2013. Hypertension in autosomal-dominant polycystic kidney disease (ADPKD) |pdf=|usr=}}{{26064509}} gab.com Text Hypertension in autosomal-dominant polycystic kidney disease (ADPKD) JO: Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26064509 #FOAvip Cardiovascular (CV) complications are the major cause of death in autosomal-dominant polycystic kidney disease (ADPKD) patients. Hypertension is common in these patients even before the onset of renal insufficiency. Blood pressure (BP) elevation is a key factor in patient outcome, mainly owing to the high prevalence of target organ damage together with a poor renal prognosis when BP is increased. Many factors have been implicated in the pathogenesis of hypertension, including the renin?angiotensin?aldosterone system (RAAS) stimulation. Polycystin deficiency may also contribute to hypertension because of its potential role in regulating the vascular tone. Early diagnosis and treatment of hypertension improve the CV and renal complications of this population. Ambulatory BP monitoring is recommended for prompt diagnosis of hypertension. CV risk assessment is mandatory. Even though a nonpharmacological approach should not be neglected, RAAS inhibitors are the cornerstone of hypertension treatment. Calcium channel blockers (CCBs) should be avoided unless resistant hypertension is present. The BP should be <140/90 mmHg in all ADPKD patients and a more intensive control (<135/85 mmHg) should be pursued as soon as microalbuminuria or left ventricle hypertrophy is present. Twit Text FOAVip Hypertension in autosomal-dominant polycystic kidney disease (ADPKD) ????Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26064509 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26064509 #FOAvip English Wikipedia <ref>{{Cite pmid|26064509}}</ref> Hypertension in autosomal-dominant polycystic kidney disease (ADPKD) #MMPMID26064509 Sans-Atxer L; Torra R; Fernández-Llama P Clin Kidney J 2013[Oct]; 6 (5): 457-63 PMID26064509show ga Cardiovascular (CV) complications are the major cause of death in autosomal-dominant polycystic kidney disease (ADPKD) patients. Hypertension is common in these patients even before the onset of renal insufficiency. Blood pressure (BP) elevation is a key factor in patient outcome, mainly owing to the high prevalence of target organ damage together with a poor renal prognosis when BP is increased. Many factors have been implicated in the pathogenesis of hypertension, including the renin?angiotensin?aldosterone system (RAAS) stimulation. Polycystin deficiency may also contribute to hypertension because of its potential role in regulating the vascular tone. Early diagnosis and treatment of hypertension improve the CV and renal complications of this population. Ambulatory BP monitoring is recommended for prompt diagnosis of hypertension. CV risk assessment is mandatory. Even though a nonpharmacological approach should not be neglected, RAAS inhibitors are the cornerstone of hypertension treatment. Calcium channel blockers (CCBs) should be avoided unless resistant hypertension is present. The BP should be <140/90 mmHg in all ADPKD patients and a more intensive control (<135/85 mmHg) should be pursued as soon as microalbuminuria or left ventricle hypertrophy is present. äHypertension in autosomal-dominant polycystic kidney disease (ADPKD) (epmc).pdf DeepDyve Pubget Overpricing