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10.1128/AAC.04781-14 http://scihub22266oqcxt.onion/10.1128/AAC.04781-14 C4432147!4432147!25870072     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Antimicrob+Agents+Chemother 2015 ; 59 (6): 3683-6 Nephropedia Template TP {{tp|p=25870072|t=2015. In Vivo Cluster Formation of Nisin and Lipid II Is Correlated with Membrane Depolarization |pdf=|usr=}}{{25870072}} gab.com Text In Vivo Cluster Formation of Nisin and Lipid II Is Correlated with Membrane Depolarization JO: Antimicrob Agents Chemother http://www.kidney.de/pget.php?&tw=1&pmid=25870072 #FOAvip Nisin and related lantibiotics kill bacteria by pore formation or by sequestering lipid II. Some lantibiotics sequester lipid II into clusters, which were suggested to kill cells through delocalized peptidoglycan synthesis. Here, we show that cluster formation is always concomitant with (i) membrane pore formation and (ii) membrane depolarization. Nisin variants that cluster lipid II kill L-form bacteria with similar efficiency, suggesting that delocalization of peptidoglycan synthesis is not the primary killing mechanism of these lantibiotics. Twit Text FOAVip In Vivo Cluster Formation of Nisin and Lipid II Is Correlated with Membrane Depolarization ????Antimicrob Agents Chemother http://www.kidney.de/pget.php?&tw=1&pmid=25870072 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25870072 #FOAvip English Wikipedia <ref>{{Cite pmid|25870072}}</ref> In Vivo Cluster Formation of Nisin and Lipid II Is Correlated with Membrane Depolarization #MMPMID25870072 Tol MB; Morales Angeles D; Scheffers DJ Antimicrob Agents Chemother 2015[Jun]; 59 (6): 3683-6 PMID25870072show ga Nisin and related lantibiotics kill bacteria by pore formation or by sequestering lipid II. Some lantibiotics sequester lipid II into clusters, which were suggested to kill cells through delocalized peptidoglycan synthesis. Here, we show that cluster formation is always concomitant with (i) membrane pore formation and (ii) membrane depolarization. Nisin variants that cluster lipid II kill L-form bacteria with similar efficiency, suggesting that delocalization of peptidoglycan synthesis is not the primary killing mechanism of these lantibiotics. äIn Vivo Cluster Formation of Nisin and Lipid II Is Correlated with Mem(epmc).pdf DeepDyve Pubget Overpricing