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10.1155/2015/269610 http://scihub22266oqcxt.onion/10.1155/2015/269610 C4430659!4430659!26064998     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Immunol+Res 2015 ; 2015 (ä): ä Nephropedia Template TP {{tp|p=26064998|t=2015. An Animal Model Using Metallic Ions to Produce Autoimmune Nephritis |pdf=|usr=}}{{26064998}} gab.com Text An Animal Model Using Metallic Ions to Produce Autoimmune Nephritis JO: J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=26064998 #FOAvip Autoimmune nephritis triggered by metallic ions was assessed in a Long-Evans rat model. The parameters evaluated included antinuclear autoantibody production, kidney damage mediated by immune complexes detected by immunofluorescence, and renal function tested by retention of nitrogen waste products and proteinuria. To accomplish our goal, the animals were treated with the following ionic metals: HgCl2, CuSO4, AgNO3, and Pb(NO3)2. A group without ionic metals was used as the control. The results of the present investigation demonstrated that metallic ions triggered antinuclear antibody production in 60% of animals, some of them with anti-DNA specificity. Furthermore, all animals treated with heavy metals developed toxic glomerulonephritis with immune complex deposition along the mesangium and membranes. These phenomena were accompanied by proteinuria and increased concentrations of urea. Based on these results, we conclude that metallic ions may induce experimental autoimmune nephritis. Twit Text FOAVip An Animal Model Using Metallic Ions to Produce Autoimmune Nephritis ????J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=26064998 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26064998 #FOAvip English Wikipedia <ref>{{Cite pmid|26064998}}</ref> An Animal Model Using Metallic Ions to Produce Autoimmune Nephritis #MMPMID26064998 Ramírez-Sandoval R; Luévano-Rodríguez N; Rodríguez-Rodríguez M; Pérez-Pérez ME; Saldívar-Elias S; Gurrola-Carlos R; Avalos-Díaz E; Bollain-y-Goytia JJ; Herrera-Esparza R J Immunol Res 2015[]; 2015 (ä): ä PMID26064998show ga Autoimmune nephritis triggered by metallic ions was assessed in a Long-Evans rat model. The parameters evaluated included antinuclear autoantibody production, kidney damage mediated by immune complexes detected by immunofluorescence, and renal function tested by retention of nitrogen waste products and proteinuria. To accomplish our goal, the animals were treated with the following ionic metals: HgCl2, CuSO4, AgNO3, and Pb(NO3)2. A group without ionic metals was used as the control. The results of the present investigation demonstrated that metallic ions triggered antinuclear antibody production in 60% of animals, some of them with anti-DNA specificity. Furthermore, all animals treated with heavy metals developed toxic glomerulonephritis with immune complex deposition along the mesangium and membranes. These phenomena were accompanied by proteinuria and increased concentrations of urea. Based on these results, we conclude that metallic ions may induce experimental autoimmune nephritis. äAn Animal Model Using Metallic Ions to Produce Autoimmune Nephritis (epmc).pdf DeepDyve Pubget Overpricing