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10.1016/j.jceh.2014.12.008 http://scihub22266oqcxt.onion/10.1016/j.jceh.2014.12.008 C4415192!4415192!25941431     free     free     free       J+Clin+Exp+Hepatol 2015 ; 5 (1): 22-40 Nephropedia Template TP {{tp|p=25941431|t=2015. Portal Vein Thrombosis |pdf=|usr=}}{{25941431}} gab.com Text Portal Vein Thrombosis JO: J Clin Exp Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=25941431 #FOAvip Portal vein thrombosis is an important cause of portal hypertension. PVT occurs in association with cirrhosis or as a result of malignant invasion by hepatocellular carcinoma or even in the absence of associated liver disease. With the current research into its genesis, majority now have an underlying prothrombotic state detectable. Endothelial activation and stagnant portal blood flow also contribute to formation of the thrombus. Acute non-cirrhotic PVT, chronic PVT (EHPVO), and portal vein thrombosis in cirrhosis are the three main variants of portal vein thrombosis with varying etiological factors and variability in presentation and management. Procoagulant state should be actively investigated. Anticoagulation is the mainstay of therapy for acute non-cirrhotic PVT, with supporting evidence for its use in cirrhotic population as well. Chronic PVT (EHPVO) on the other hand requires the management of portal hypertension as such and with role for anticoagulation in the setting of underlying prothrombotic state, however data is awaited in those with no underlying prothrombotic states. TIPS and liver transplant may be feasible even in the setting of PVT however proper selection of candidates and type of surgery is warranted. Thrombolysis and thrombectomy have some role. TARE is a new modality for management of HCC with portal vein invasion. Twit Text FOAVip Portal Vein Thrombosis ????J Clin Exp Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=25941431 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25941431 #FOAvip English Wikipedia <ref>{{Cite pmid|25941431}}</ref> Portal Vein Thrombosis #MMPMID25941431 Chawla YK; Bodh V J Clin Exp Hepatol 2015[Mar]; 5 (1): 22-40 PMID25941431show ga Portal vein thrombosis is an important cause of portal hypertension. PVT occurs in association with cirrhosis or as a result of malignant invasion by hepatocellular carcinoma or even in the absence of associated liver disease. With the current research into its genesis, majority now have an underlying prothrombotic state detectable. Endothelial activation and stagnant portal blood flow also contribute to formation of the thrombus. Acute non-cirrhotic PVT, chronic PVT (EHPVO), and portal vein thrombosis in cirrhosis are the three main variants of portal vein thrombosis with varying etiological factors and variability in presentation and management. Procoagulant state should be actively investigated. Anticoagulation is the mainstay of therapy for acute non-cirrhotic PVT, with supporting evidence for its use in cirrhotic population as well. Chronic PVT (EHPVO) on the other hand requires the management of portal hypertension as such and with role for anticoagulation in the setting of underlying prothrombotic state, however data is awaited in those with no underlying prothrombotic states. TIPS and liver transplant may be feasible even in the setting of PVT however proper selection of candidates and type of surgery is warranted. Thrombolysis and thrombectomy have some role. TARE is a new modality for management of HCC with portal vein invasion. äPortal Vein Thrombosis (epmc).pdf DeepDyve Pubget Overpricing