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10.1093/ckj/sfs026 http://scihub22266oqcxt.onion/10.1093/ckj/sfs026 C4400504!4400504!26069767     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Kidney+J 2012 ; 5 (3): 217-20 Nephropedia Template TP {{tp|p=26069767|t=2012. Paradoxical hypertension and salt wasting in Type II Bartter syndrome |pdf=|usr=}}{{26069767}} gab.com Text Paradoxical hypertension and salt wasting in Type II Bartter syndrome JO: Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26069767 #FOAvip Ante/neonatal Bartter syndrome (BS) is a rare hereditary disorder. It is characterized by renal salt wasting, hypokalaemic metabolic alkalosis, high renin and aldosterone but normal blood pressure. We report a low birth weight newborn baby who presented with repeated apnoea shortly after birth as well as hyponatraemia, hypochloraemia, hyperkalaemia and metabolic acidosis. Her biochemical features mimicked pseudohypoaldosteronism but with initial hypertension, which had not been described in BS. Her subsequent genetic study confirmed two novel heterozygous mutations in the Exon 5 of KCNJ1 compatible with Type II BS. Twit Text FOAVip Paradoxical hypertension and salt wasting in Type II Bartter syndrome ????Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=26069767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26069767 #FOAvip English Wikipedia <ref>{{Cite pmid|26069767}}</ref> Paradoxical hypertension and salt wasting in Type II Bartter syndrome #MMPMID26069767 Chan WKy; To KF; Tong JHM; Law CW Clin Kidney J 2012[Jun]; 5 (3): 217-20 PMID26069767show ga Ante/neonatal Bartter syndrome (BS) is a rare hereditary disorder. It is characterized by renal salt wasting, hypokalaemic metabolic alkalosis, high renin and aldosterone but normal blood pressure. We report a low birth weight newborn baby who presented with repeated apnoea shortly after birth as well as hyponatraemia, hypochloraemia, hyperkalaemia and metabolic acidosis. Her biochemical features mimicked pseudohypoaldosteronism but with initial hypertension, which had not been described in BS. Her subsequent genetic study confirmed two novel heterozygous mutations in the Exon 5 of KCNJ1 compatible with Type II BS. äParadoxical hypertension and salt wasting in Type II Bartter syndrome (epmc).pdf DeepDyve Pubget Overpricing