Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1007/s11926-014-0472-1 http://scihub22266oqcxt.onion/10.1007/s11926-014-0472-1 C4394866!4394866!25475595     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Rheumatol+Rep 2015 ; 17 (1): 472 Nephropedia Template TP {{tp|p=25475595|t=2015. The Role of the Gut Microbiota in the Pathogenesis of Antiphospholipid Syndrome |pdf=|usr=}}{{25475595}} gab.com Text The Role of the Gut Microbiota in the Pathogenesis of Antiphospholipid Syndrome JO: Curr Rheumatol Rep http://www.kidney.de/pget.php?&tw=1&pmid=25475595 #FOAvip Infectious triggers are associated with the induction of transient antiphospholipid antibodies. One therefore wonders if microbes that permanently colonize us play a role in the pathogenesis of antiphospholipid syndrome (APS). The microbiota represents the collection of all microorganisms colonizing humans and is necessary for normal host physiology. The microbiota, however, is a constant stress on the immune system, which is tasked with recognizing and eliminating pathogenic microbes while tolerating commensal populations. A growing body of literature supports a critical role for the commensal-immune axis in the development of autoimmunity against colonized barriers (e.g., gut or skin) and sterile organs (e.g., pancreas or joints). Whether these interactions affect the development and sustainment of autoreactive CD4+ T cells and pathogenic autoantibodies in APS is unknown. This review provides an overview of the current understanding of the commensal-immune axis in autoimmunity with a focus on the potential relevance to APS. Additionally, we discuss emerging findings supporting the involvement of the gut microbiota in a spontaneous model of APS, the (NZW×BXSB)F1 hybrid, and formalize hypotheses to explain how interactions between the immune system and the microbiota may influence human APS etiopathogenesis. Twit Text FOAVip The Role of the Gut Microbiota in the Pathogenesis of Antiphospholipid Syndrome ????Curr Rheumatol Rep http://www.kidney.de/pget.php?&tw=1&pmid=25475595 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25475595 #FOAvip English Wikipedia <ref>{{Cite pmid|25475595}}</ref> The Role of the Gut Microbiota in the Pathogenesis of Antiphospholipid Syndrome #MMPMID25475595 Ruff WE; Vieira SM; Kriegel MA Curr Rheumatol Rep 2015[Jan]; 17 (1): 472 PMID25475595show ga Infectious triggers are associated with the induction of transient antiphospholipid antibodies. One therefore wonders if microbes that permanently colonize us play a role in the pathogenesis of antiphospholipid syndrome (APS). The microbiota represents the collection of all microorganisms colonizing humans and is necessary for normal host physiology. The microbiota, however, is a constant stress on the immune system, which is tasked with recognizing and eliminating pathogenic microbes while tolerating commensal populations. A growing body of literature supports a critical role for the commensal-immune axis in the development of autoimmunity against colonized barriers (e.g., gut or skin) and sterile organs (e.g., pancreas or joints). Whether these interactions affect the development and sustainment of autoreactive CD4+ T cells and pathogenic autoantibodies in APS is unknown. This review provides an overview of the current understanding of the commensal-immune axis in autoimmunity with a focus on the potential relevance to APS. Additionally, we discuss emerging findings supporting the involvement of the gut microbiota in a spontaneous model of APS, the (NZW×BXSB)F1 hybrid, and formalize hypotheses to explain how interactions between the immune system and the microbiota may influence human APS etiopathogenesis. äThe Role of the Gut Microbiota in the Pathogenesis of Antiphospholipid(epmc).pdf DeepDyve Pubget Overpricing