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10.1155/2015/314178 http://scihub22266oqcxt.onion/10.1155/2015/314178 C4389985!4389985 !25883953     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25883953 &cmd=llinks): Failed to open stream: HTTP request failed! 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Actin-tethered junctional complexes in angiogenesis and lymphangiogenesis in association with vascular endothelial growth factor |pdf=|usr=}}{{25883953 }} gab.com Text Actin-tethered junctional complexes in angiogenesis and lymphangiogenesis in association with vascular endothelial growth factor JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=25883953 #FOAvip Vasculature is present in all tissues and therefore is indispensable for development, biology, and pathology of multicellular organisms. Endothelial cells guarantee proper function of the vessels and are the original component in angiogenesis. Morphogenesis of the vascular system utilizes processes like cell adhesion, motility, proliferation, and survival that are closely related to the dynamics of actin filaments and actin-tethered adhesion complexes. Here we review involvement of actin cytoskeleton-associated junctional molecules of endothelial cells in angiogenesis and lymphangiogenesis. Particularly, we focus on F-actin binding protein afadin, an adaptor protein involved in broad range of signaling mechanisms. Afadin mediates the pathways of vascular endothelial growth factor- (VEGF-) and sphingosine 1-phosphate-triggered angiogenesis and is essential for embryonic development of lymph vessels in mice. We propose that targeting actin-tethered junctional molecules, including afadin, may present a new approach to angiogenic therapy that in combination with today used medications like VEGF inhibitors will benefit against development of pathological angiogenesis. Twit Text FOAVip Actin-tethered junctional complexes in angiogenesis and lymphangiogenesis in association with vascular endothelial growth factor ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=25883953 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25883953 #FOAvip English Wikipedia <ref>{{Cite pmid|25883953 }}</ref> Actin-tethered junctional complexes in angiogenesis and lymphangiogenesis in association with vascular endothelial growth factor #MMPMID25883953 Zankov DP ; Ogita H Biomed Res Int 2015[]; 2015 (ä): 314178 PMID25883953 show ga Vasculature is present in all tissues and therefore is indispensable for development, biology, and pathology of multicellular organisms. Endothelial cells guarantee proper function of the vessels and are the original component in angiogenesis. Morphogenesis of the vascular system utilizes processes like cell adhesion, motility, proliferation, and survival that are closely related to the dynamics of actin filaments and actin-tethered adhesion complexes. Here we review involvement of actin cytoskeleton-associated junctional molecules of endothelial cells in angiogenesis and lymphangiogenesis. Particularly, we focus on F-actin binding protein afadin, an adaptor protein involved in broad range of signaling mechanisms. Afadin mediates the pathways of vascular endothelial growth factor- (VEGF-) and sphingosine 1-phosphate-triggered angiogenesis and is essential for embryonic development of lymph vessels in mice. We propose that targeting actin-tethered junctional molecules, including afadin, may present a new approach to angiogenic therapy that in combination with today used medications like VEGF inhibitors will benefit against development of pathological angiogenesis. |*Gene Expression Regulation [MESH] |*Lymphangiogenesis [MESH] |*Neovascularization, Pathologic [MESH] |Actins/*physiology [MESH] |Animals [MESH] |Antigens, CD/metabolism [MESH] |Cadherins/metabolism [MESH] |Catenins/metabolism [MESH] |Cytoskeleton/metabolism [MESH] |Delta Catenin [MESH] |Endothelial Cells/metabolism [MESH] |Humans [MESH] |Integrins/metabolism [MESH] |Lymphatic Vessels/pathology [MESH] |Lysophospholipids/metabolism [MESH] |Mice [MESH] |Mice, Knockout [MESH] |Microfilament Proteins/*physiology [MESH] |Neovascularization, Physiologic [MESH] |Sphingosine/analogs & derivatives/metabolism [MESH] |Vascular Endothelial Growth Factor A/*metabolism [MESH]Actin-tethered junctional complexes in angiogenesis and lymphangiogene(epmc).pdf DeepDyve Pubget Overpricing