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10.1113/jphysiol.2014.281204 http://scihub22266oqcxt.onion/10.1113/jphysiol.2014.281204 C4376414!4376414!25772289     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25772289&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Physiol 2015 ; 593 (Pt 6): 1331-45 Nephropedia Template TP {{tp|p=25772289|t=2015. Deranged sodium to sudden death |pdf=|usr=}}{{25772289}} gab.com Text Deranged sodium to sudden death JO: J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=25772289 #FOAvip In February 2014, a group of scientists convened as part of the University of California Davis Cardiovascular Symposium to bring together experimental and mathematical modelling perspectives and discuss points of consensus and controversy on the topic of sodium in the heart. This paper summarizes the topics of presentation and discussion from the symposium, with a focus on the role of aberrant sodium channels and abnormal sodium homeostasis in cardiac arrhythmias and pharmacotherapy from the subcellular scale to the whole heart. Two following papers focus on Na+ channel structure, function and regulation, and Na+/Ca2+ exchange and Na+/K+ ATPase. The UC Davis Cardiovascular Symposium is a biannual event that aims to bring together leading experts in subfields of cardiovascular biomedicine to focus on topics of importance to the field. The focus on Na+ in the 2014 symposium stemmed from the multitude of recent studies that point to the importance of maintaining Na+ homeostasis in the heart, as disruption of homeostatic processes are increasingly identified in cardiac disease states. Understanding how disruption in cardiac Na+-based processes leads to derangement in multiple cardiac components at the level of the cell and to then connect these perturbations to emergent behaviour in the heart to cause disease is a critical area of research. The ubiquity of disruption of Na+ channels and Na+ homeostasis in cardiac disorders of excitability and mechanics emphasizes the importance of a fundamental understanding of the associated mechanisms and disease processes to ultimately reveal new targets for human therapy. Twit Text FOAVip Deranged sodium to sudden death ????J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=25772289 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25772289 #FOAvip English Wikipedia <ref>{{Cite pmid|25772289}}</ref> Deranged sodium to sudden death #MMPMID25772289 Clancy CE; Chen-Izu Y; Bers DM; Belardinelli L; Boyden PA; Csernoch L; Despa S; Fermini B; Hool LC; Izu L; Kass RS; Lederer WJ; Louch WE; Maack C; Matiazzi A; Qu Z; Rajamani S; Rippinger CM; Sejersted OM; O'Rourke B; Weiss JN; Varró A; Zaza A J Physiol 2015[Mar]; 593 (Pt 6): 1331-45 PMID25772289show ga In February 2014, a group of scientists convened as part of the University of California Davis Cardiovascular Symposium to bring together experimental and mathematical modelling perspectives and discuss points of consensus and controversy on the topic of sodium in the heart. This paper summarizes the topics of presentation and discussion from the symposium, with a focus on the role of aberrant sodium channels and abnormal sodium homeostasis in cardiac arrhythmias and pharmacotherapy from the subcellular scale to the whole heart. Two following papers focus on Na+ channel structure, function and regulation, and Na+/Ca2+ exchange and Na+/K+ ATPase. The UC Davis Cardiovascular Symposium is a biannual event that aims to bring together leading experts in subfields of cardiovascular biomedicine to focus on topics of importance to the field. The focus on Na+ in the 2014 symposium stemmed from the multitude of recent studies that point to the importance of maintaining Na+ homeostasis in the heart, as disruption of homeostatic processes are increasingly identified in cardiac disease states. Understanding how disruption in cardiac Na+-based processes leads to derangement in multiple cardiac components at the level of the cell and to then connect these perturbations to emergent behaviour in the heart to cause disease is a critical area of research. The ubiquity of disruption of Na+ channels and Na+ homeostasis in cardiac disorders of excitability and mechanics emphasizes the importance of a fundamental understanding of the associated mechanisms and disease processes to ultimately reveal new targets for human therapy. äDeranged sodium to sudden death (epmc).pdf DeepDyve Pubget Overpricing