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10.3389/fonc.2015.00063 http://scihub22266oqcxt.onion/10.3389/fonc.2015.00063 C4369728!4369728!25853091     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Oncol 2015 ; 5 (ä): ä Nephropedia Template TP {{tp|p=25853091|t=2015. Cancer-Associated Fibroblasts Connect Metastasis-Promoting Communication in Colorectal Cancer |pdf=|usr=}}{{25853091}} gab.com Text Cancer-Associated Fibroblasts Connect Metastasis-Promoting Communication in Colorectal Cancer JO: Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=25853091 #FOAvip Colorectal cancer (CRC) progression and eventually metastasis is directed in many aspects by a circuitous ecosystem consisting of an extracellular matrix scaffold populated by cancer-associated fibroblasts (CAFs), endothelial cells, and diverse immune cells. CAFs are recruited from local tissue-resident fibroblasts or pericryptal fibroblasts and distant fibroblast precursors. CAFs are highly abundant in CRC. In this review, we apply the metastasis-promoting communication of colorectal CAFs to 10 cancer hallmarks described by Hanahan and Weinberg. CAFs influence innate and adaptive tumor immune responses. Using datasets from previously published work, we re-explore the potential messages implicated in this process. Fibroblasts present in metastasis (metastasis-associated fibroblasts) from CRC may have other characteristics and functional roles than CAFs in the primary tumor. Since CAFs connect metastasis-promoting communication, CAF markers are potential prognostic biomarkers. CAFs and their products are possible targets for novel therapeutic strategies. Twit Text FOAVip Cancer-Associated Fibroblasts Connect Metastasis-Promoting Communication in Colorectal Cancer ????Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=25853091 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25853091 #FOAvip English Wikipedia <ref>{{Cite pmid|25853091}}</ref> Cancer-Associated Fibroblasts Connect Metastasis-Promoting Communication in Colorectal Cancer #MMPMID25853091 Tommelein J; Verset L; Boterberg T; Demetter P; Bracke M; De Wever O Front Oncol 2015[]; 5 (ä): ä PMID25853091show ga Colorectal cancer (CRC) progression and eventually metastasis is directed in many aspects by a circuitous ecosystem consisting of an extracellular matrix scaffold populated by cancer-associated fibroblasts (CAFs), endothelial cells, and diverse immune cells. CAFs are recruited from local tissue-resident fibroblasts or pericryptal fibroblasts and distant fibroblast precursors. CAFs are highly abundant in CRC. In this review, we apply the metastasis-promoting communication of colorectal CAFs to 10 cancer hallmarks described by Hanahan and Weinberg. CAFs influence innate and adaptive tumor immune responses. Using datasets from previously published work, we re-explore the potential messages implicated in this process. Fibroblasts present in metastasis (metastasis-associated fibroblasts) from CRC may have other characteristics and functional roles than CAFs in the primary tumor. Since CAFs connect metastasis-promoting communication, CAF markers are potential prognostic biomarkers. CAFs and their products are possible targets for novel therapeutic strategies. äCancer-Associated Fibroblasts Connect Metastasis-Promoting Communicati(epmc).pdf DeepDyve Pubget Overpricing