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10.1016/j.mito.2015.01.009 http://scihub22266oqcxt.onion/10.1016/j.mito.2015.01.009 C4364530!4364530 !25660179     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25660179 &cmd=llinks): Failed to open stream: HTTP request failed! 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Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors |pdf=|usr=}}{{25660179 }} gab.com Text Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors JO: Mitochondrion http://www.kidney.de/pget.php?&tw=1&pmid=25660179 #FOAvip Mitochondrial disorders are the most common inborn errors of metabolism affecting the oxidative phosphorylation system (OXPHOS). Because of the poor knowledge of the pathogenic mechanisms, a cure for these disorders is still unavailable and all the treatments currently in use are supportive more than curative. Therefore, in the past decade a great variety of mouse models have been developed to assess the in vivo function of several mitochondrial proteins involved in human diseases. Due to the genetic and physiological similarity to humans, mice represent reliable models to study the pathogenic mechanisms of mitochondrial disorders and are precious to test new therapeutic approaches. Here we summarize the features of several mouse models of mitochondrial diseases directly related to defects in subunits of the OXPHOS complexes or in assembly factors. We discuss how these models recapitulate many human conditions and how they have contributed to the understanding of mitochondrial function in health and disease. Twit Text FOAVip Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors ????Mitochondrion http://www.kidney.de/pget.php?&tw=1&pmid=25660179 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25660179 #FOAvip English Wikipedia <ref>{{Cite pmid|25660179 }}</ref> Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors #MMPMID25660179 Torraco A ; Peralta S ; Iommarini L ; Diaz F Mitochondrion 2015[Mar]; 21 (ä): 76-91 PMID25660179 show ga Mitochondrial disorders are the most common inborn errors of metabolism affecting the oxidative phosphorylation system (OXPHOS). Because of the poor knowledge of the pathogenic mechanisms, a cure for these disorders is still unavailable and all the treatments currently in use are supportive more than curative. Therefore, in the past decade a great variety of mouse models have been developed to assess the in vivo function of several mitochondrial proteins involved in human diseases. Due to the genetic and physiological similarity to humans, mice represent reliable models to study the pathogenic mechanisms of mitochondrial disorders and are precious to test new therapeutic approaches. Here we summarize the features of several mouse models of mitochondrial diseases directly related to defects in subunits of the OXPHOS complexes or in assembly factors. We discuss how these models recapitulate many human conditions and how they have contributed to the understanding of mitochondrial function in health and disease. |*Disease Models, Animal [MESH] |*Oxidative Phosphorylation [MESH] |Animals [MESH] |Humans [MESH] |Mice [MESH] |Mitochondrial Diseases/*physiopathology [MESH]Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies cau(epmc).pdf DeepDyve Pubget Overpricing