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10.1016/j.ymgme.2014.11.017 http://scihub22266oqcxt.onion/10.1016/j.ymgme.2014.11.017 C4355162!4355162!25533111     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Mol+Genet+Metab 2015 ; 114 (3): 397-402 Nephropedia Template TP {{tp|p=25533111|t=2015. Targeting Novel Signaling Pathways for Resistant Acute Myeloid Leukemia |pdf=|usr=}}{{25533111}} gab.com Text Targeting Novel Signaling Pathways for Resistant Acute Myeloid Leukemia JO: Mol Genet Metab http://www.kidney.de/pget.php?&tw=1&pmid=25533111 #FOAvip Acute myeloid leukemia (AML) is a hematologic malignancy that is the most common type of acute leukemia diagnosed in adults and the second most common type in children. The overall survival is poor and treatment is associated with significant complications and even death. In addition, a significant number of patients will not respond to therapy or relapse. In this review, several new signaling proteins aberrantly regulated in AML are described, including CREB, Triad1, Bcl-2 family members, Stat3, and mTOR/MEK. Identifying more effective and less toxic agents will provide novel approaches to treat AML. Twit Text FOAVip Targeting Novel Signaling Pathways for Resistant Acute Myeloid Leukemia ????Mol Genet Metab http://www.kidney.de/pget.php?&tw=1&pmid=25533111 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25533111 #FOAvip English Wikipedia <ref>{{Cite pmid|25533111}}</ref> Targeting Novel Signaling Pathways for Resistant Acute Myeloid Leukemia #MMPMID25533111 Sakamoto KM; Grant S; Saleiro D; Crispino J; Hijiya N; Giles F; Platanias L; Eklund EA Mol Genet Metab 2015[Mar]; 114 (3): 397-402 PMID25533111show ga Acute myeloid leukemia (AML) is a hematologic malignancy that is the most common type of acute leukemia diagnosed in adults and the second most common type in children. The overall survival is poor and treatment is associated with significant complications and even death. In addition, a significant number of patients will not respond to therapy or relapse. In this review, several new signaling proteins aberrantly regulated in AML are described, including CREB, Triad1, Bcl-2 family members, Stat3, and mTOR/MEK. Identifying more effective and less toxic agents will provide novel approaches to treat AML. äTargeting Novel Signaling Pathways for Resistant Acute Myeloid Leukemi(epmc).pdf DeepDyve Pubget Overpricing