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10.3390/ijms16024083 http://scihub22266oqcxt.onion/10.3390/ijms16024083 C4346945!4346945!25689426     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2015 ; 16 (2): 4083-94 Nephropedia Template TP {{tp|p=25689426|t=2015. Autophagy as a Regulatory Component of Erythropoiesis |pdf=|usr=}}{{25689426}} gab.com Text Autophagy as a Regulatory Component of Erythropoiesis JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=25689426 #FOAvip Autophagy is a process that leads to the degradation of unnecessary or dysfunctional cellular components and long-lived protein aggregates. Erythropoiesis is a branch of hematopoietic differentiation by which mature red blood cells (RBCs) are generated from multi-potential hematopoietic stem cells (HSCs). Autophagy plays a critical role in the elimination of mitochondria, ribosomes and other organelles during erythroid terminal differentiation. Here, the modulators of autophagy that regulate erythroid differentiation were summarized, including autophagy-related (Atg) genes, the B-cell lymphoma 2 (Bcl-2) family member Bcl-2/adenovirus E1B 19 kDa interacting protein 3-like (Nix/Binp3L), transcription factors globin transcription factor 1 (GATA1) and forkhead box O3 (FoxO3), intermediary factor KRAB-associated protein1 (KAP1), and other modulators, such as focal adhesion kinase family-interacting protein of 200-kDa (FIP200), Ca2+ and 15-lipoxygenase. Understanding the modulators of autophagy in erythropoiesis will benefit the autophagy research field and facilitate the prevention and treatment of autophagy-related red blood cell disorders. Twit Text FOAVip Autophagy as a Regulatory Component of Erythropoiesis ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=25689426 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25689426 #FOAvip English Wikipedia <ref>{{Cite pmid|25689426}}</ref> Autophagy as a Regulatory Component of Erythropoiesis #MMPMID25689426 Zhang J; Wu K; Xiao X; Liao J; Hu Q; Chen H; Liu J; An X Int J Mol Sci 2015[Feb]; 16 (2): 4083-94 PMID25689426show ga Autophagy is a process that leads to the degradation of unnecessary or dysfunctional cellular components and long-lived protein aggregates. Erythropoiesis is a branch of hematopoietic differentiation by which mature red blood cells (RBCs) are generated from multi-potential hematopoietic stem cells (HSCs). Autophagy plays a critical role in the elimination of mitochondria, ribosomes and other organelles during erythroid terminal differentiation. Here, the modulators of autophagy that regulate erythroid differentiation were summarized, including autophagy-related (Atg) genes, the B-cell lymphoma 2 (Bcl-2) family member Bcl-2/adenovirus E1B 19 kDa interacting protein 3-like (Nix/Binp3L), transcription factors globin transcription factor 1 (GATA1) and forkhead box O3 (FoxO3), intermediary factor KRAB-associated protein1 (KAP1), and other modulators, such as focal adhesion kinase family-interacting protein of 200-kDa (FIP200), Ca2+ and 15-lipoxygenase. Understanding the modulators of autophagy in erythropoiesis will benefit the autophagy research field and facilitate the prevention and treatment of autophagy-related red blood cell disorders. äAutophagy as a Regulatory Component of Erythropoiesis (epmc).pdf DeepDyve Pubget Overpricing