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10.1016/j.chemphyslip.2013.12.009 http://scihub22266oqcxt.onion/10.1016/j.chemphyslip.2013.12.009 C4342993!4342993 !24445246     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24445246 &cmd=llinks): Failed to open stream: HTTP request failed! 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The functions of cardiolipin in cellular metabolism-potential modifiers of the Barth syndrome phenotype |pdf=|usr=}}{{24445246 }} gab.com Text The functions of cardiolipin in cellular metabolism-potential modifiers of the Barth syndrome phenotype JO: Chem Phys Lipids http://www.kidney.de/pget.php?&tw=1&pmid=24445246 #FOAvip The phospholipid cardiolipin (CL) plays a role in many cellular functions and signaling pathways both inside and outside of mitochondria. This review focuses on the role of CL in energy metabolism. Many reactions of electron transport and oxidative phosphorylation, the transport of metabolites required for these processes, and the stabilization of electron transport chain supercomplexes require CL. Recent studies indicate that CL is required for the synthesis of iron-sulfur (Fe-S) co-factors, which are essential for numerous metabolic pathways. Activation of carnitine shuttle enzymes that are required for fatty acid metabolism is CL dependent. The presence of substantial amounts of CL in the peroxisomal membrane suggests that CL may be required for peroxisomal functions. Understanding the role of CL in energy metabolism may identify physiological modifiers that exacerbate the loss of CL and underlie the variation in symptoms observed in Barth syndrome, a genetic disorder of CL metabolism. Twit Text FOAVip The functions of cardiolipin in cellular metabolism-potential modifiers of the Barth syndrome phenotype ????Chem Phys Lipids http://www.kidney.de/pget.php?&tw=1&pmid=24445246 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24445246 #FOAvip English Wikipedia <ref>{{Cite pmid|24445246 }}</ref> The functions of cardiolipin in cellular metabolism-potential modifiers of the Barth syndrome phenotype #MMPMID24445246 Raja V ; Greenberg ML Chem Phys Lipids 2014[Apr]; 179 (ä): 49-56 PMID24445246 show ga The phospholipid cardiolipin (CL) plays a role in many cellular functions and signaling pathways both inside and outside of mitochondria. This review focuses on the role of CL in energy metabolism. Many reactions of electron transport and oxidative phosphorylation, the transport of metabolites required for these processes, and the stabilization of electron transport chain supercomplexes require CL. Recent studies indicate that CL is required for the synthesis of iron-sulfur (Fe-S) co-factors, which are essential for numerous metabolic pathways. Activation of carnitine shuttle enzymes that are required for fatty acid metabolism is CL dependent. The presence of substantial amounts of CL in the peroxisomal membrane suggests that CL may be required for peroxisomal functions. Understanding the role of CL in energy metabolism may identify physiological modifiers that exacerbate the loss of CL and underlie the variation in symptoms observed in Barth syndrome, a genetic disorder of CL metabolism. |*Phenotype [MESH] |Animals [MESH] |Barth Syndrome/*metabolism/pathology [MESH] |Cardiolipins/chemistry/*metabolism [MESH] |Energy Metabolism [MESH] |Homeostasis [MESH] |Humans [MESH]The functions of cardiolipin in cellular metabolism-potential modifier(epmc).pdf DeepDyve Pubget Overpricing