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10.1681/ASN.2014050509 http://scihub22266oqcxt.onion/10.1681/ASN.2014050509 C4341488!4341488!25190731     free     free     free       J+Am+Soc+Nephrol 2015 ; 26 (3): 525-9 Nephropedia Template TP {{tp|p=25190731|t=2015. Crystalglobulin-Induced Nephropathy |pdf=|usr=}}{{25190731}} gab.com Text Crystalglobulin-Induced Nephropathy JO: J Am Soc Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25190731 #FOAvip Crystalline nephropathy refers to renal parenchymal deposition of crystals leading to kidney damage. The most common forms of crystalline nephropathy encountered in renal pathology are nephrocalcinosis and oxalate nephropathy. Less frequent types include urate nephropathy, cystinosis, dihydroxyadeninuria, and drug-induced crystalline nephropathy (e.g., caused by indinavir or triamterene). Monoclonal proteins can also deposit in the kidney as crystals and cause tissue damage. This occurs in conditions such as light chain proximal tubulopathy, crystal-storing histiocytosis, and crystalglobulinemia. The latter is a rare complication of multiple myeloma that results from crystallization of monoclonal proteins in the systemic vasculature, leading to vascular injury, thrombosis, and occlusion. In this report, we describe a case of crystalglobulin-induced nephropathy and discuss its pathophysiology and the differential diagnosis of paraprotein-induced crystalline nephropathy. Twit Text FOAVip Crystalglobulin-Induced Nephropathy ????J Am Soc Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25190731 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25190731 #FOAvip English Wikipedia <ref>{{Cite pmid|25190731}}</ref> Crystalglobulin-Induced Nephropathy #MMPMID25190731 Gupta V; El Ters M; Kashani K; Leung N; Nasr SH J Am Soc Nephrol 2015[Mar]; 26 (3): 525-9 PMID25190731show ga Crystalline nephropathy refers to renal parenchymal deposition of crystals leading to kidney damage. The most common forms of crystalline nephropathy encountered in renal pathology are nephrocalcinosis and oxalate nephropathy. Less frequent types include urate nephropathy, cystinosis, dihydroxyadeninuria, and drug-induced crystalline nephropathy (e.g., caused by indinavir or triamterene). Monoclonal proteins can also deposit in the kidney as crystals and cause tissue damage. This occurs in conditions such as light chain proximal tubulopathy, crystal-storing histiocytosis, and crystalglobulinemia. The latter is a rare complication of multiple myeloma that results from crystallization of monoclonal proteins in the systemic vasculature, leading to vascular injury, thrombosis, and occlusion. In this report, we describe a case of crystalglobulin-induced nephropathy and discuss its pathophysiology and the differential diagnosis of paraprotein-induced crystalline nephropathy. äCrystalglobulin-Induced Nephropathy (epmc).pdf DeepDyve Pubget Overpricing