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10.1016/j.tibs.2015.01.002 http://scihub22266oqcxt.onion/10.1016/j.tibs.2015.01.002 C4340752!4340752!25656104     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Trends+Biochem+Sci 2015 ; 40 (3): 141-8 Nephropedia Template TP {{tp|p=25656104|t=2015. UPR, autophagy and mitochondria crosstalk underlies the ER stress response |pdf=|usr=}}{{25656104}} gab.com Text UPR, autophagy and mitochondria crosstalk underlies the ER stress response JO: Trends Biochem Sci http://www.kidney.de/pget.php?&tw=1&pmid=25656104 #FOAvip Cellular stress, induced by external or internal cues, activates several well-orchestrated processes aimed at either restoring cellular homeostasis or committing to cell death. Those processes include the unfolded protein response (UPR), autophagy, hypoxia, and mitochondrial function, which are part of the global ER stress (ERS) response. When one of the ERS elements is impaired, as often occurs under pathological conditions, overall cellular homeostasis may be perturbed. Further, activation of the UPR could trigger changes in mitochondrial function or autophagy, which could modulate the UPR, exemplifying cross-talk processes. Among the numerous factors that control the magnitude or duration of these processes are ubiquitin ligases, which govern overall cellular stress outcomes. Here we summarize crosstalk among fundamental processes governing ERS responses. Twit Text FOAVip UPR, autophagy and mitochondria crosstalk underlies the ER stress response ????Trends Biochem Sci http://www.kidney.de/pget.php?&tw=1&pmid=25656104 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25656104 #FOAvip English Wikipedia <ref>{{Cite pmid|25656104}}</ref> UPR, autophagy and mitochondria crosstalk underlies the ER stress response #MMPMID25656104 Senft D; Ronai ZA Trends Biochem Sci 2015[Mar]; 40 (3): 141-8 PMID25656104show ga Cellular stress, induced by external or internal cues, activates several well-orchestrated processes aimed at either restoring cellular homeostasis or committing to cell death. Those processes include the unfolded protein response (UPR), autophagy, hypoxia, and mitochondrial function, which are part of the global ER stress (ERS) response. When one of the ERS elements is impaired, as often occurs under pathological conditions, overall cellular homeostasis may be perturbed. Further, activation of the UPR could trigger changes in mitochondrial function or autophagy, which could modulate the UPR, exemplifying cross-talk processes. Among the numerous factors that control the magnitude or duration of these processes are ubiquitin ligases, which govern overall cellular stress outcomes. Here we summarize crosstalk among fundamental processes governing ERS responses. äUPR, autophagy and mitochondria crosstalk underlies the ER stress resp(epmc).pdf DeepDyve Pubget Overpricing