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10.1111/imr.12256 http://scihub22266oqcxt.onion/10.1111/imr.12256 C4339221!4339221!25703569     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Immunol+Rev 2015 ; 264 (1): 319-26 Nephropedia Template TP {{tp|p=25703569|t=2015. Mycobacterial Genes Essential for the Pathogen s Survival in the Host |pdf=|usr=}}{{25703569}} gab.com Text Mycobacterial Genes Essential for the Pathogen s Survival in the Host JO: Immunol Rev http://www.kidney.de/pget.php?&tw=1&pmid=25703569 #FOAvip Mycobacterium tuberculosis (Mtb) has evolved within the human immune system as both host and reservoir. The study of genes required for its growth and persistence in vivo thus offers linked insights into its pathogenicity and host immunity. Studies of Mtb mutants have implicated metabolic adaptation (consisting of carbon, nitrogen, vitamin and cofactor metabolism), intrabacterial pH homeostasis, and defense against reactive oxygen and reactive nitrogen species, as key determinants of its pathogenicity. However, the mechanisms of host immunity are complex and often combinatorial. Growing evidence has thus begun to reveal that the determinants of Mtb's pathogenicity may serve a broader and more complex array of functions than the isolated experimental settings in which they were initially found. Here, we review select examples, which exemplify this complexity, highlighting the distinct phases of Mtb's life cycle and the diverse microenvironments encountered therein. Twit Text FOAVip Mycobacterial Genes Essential for the Pathogen s Survival in the Host ????Immunol Rev http://www.kidney.de/pget.php?&tw=1&pmid=25703569 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25703569 #FOAvip English Wikipedia <ref>{{Cite pmid|25703569}}</ref> Mycobacterial Genes Essential for the Pathogen s Survival in the Host #MMPMID25703569 Ehrt S; Rhee K; Schnappinger D Immunol Rev 2015[Mar]; 264 (1): 319-26 PMID25703569show ga Mycobacterium tuberculosis (Mtb) has evolved within the human immune system as both host and reservoir. The study of genes required for its growth and persistence in vivo thus offers linked insights into its pathogenicity and host immunity. Studies of Mtb mutants have implicated metabolic adaptation (consisting of carbon, nitrogen, vitamin and cofactor metabolism), intrabacterial pH homeostasis, and defense against reactive oxygen and reactive nitrogen species, as key determinants of its pathogenicity. However, the mechanisms of host immunity are complex and often combinatorial. Growing evidence has thus begun to reveal that the determinants of Mtb's pathogenicity may serve a broader and more complex array of functions than the isolated experimental settings in which they were initially found. Here, we review select examples, which exemplify this complexity, highlighting the distinct phases of Mtb's life cycle and the diverse microenvironments encountered therein. äMycobacterial Genes Essential for the Pathogen s Survival in the Host (epmc).pdf DeepDyve Pubget Overpricing