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10.1016/j.autneu.2014.10.014 http://scihub22266oqcxt.onion/10.1016/j.autneu.2014.10.014 C4336605!4336605!25458431     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25458431&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Auton+Neurosci 2015 ; ä (ä): 69-73 Nephropedia Template TP {{tp|p=25458431|t=2015. The exercise pressor reflex and peripheral artery disease |pdf=|usr=}}{{25458431}} gab.com Text The exercise pressor reflex and peripheral artery disease JO: Auton Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25458431 #FOAvip The exercise pressor reflex contributes to increases in cardiovascular and ventilatory function during exercise. These reflexive increases are caused by both mechanical and metabolic stimulation of Group III and IV afferents with endings in contracting skeletal muscle. Patients with peripheral artery disease (PAD) have an augmented exercise pressor reflex. Recently, an animal model of PAD was established which allows further investigation of possible mechanisms involved in this augmented reflex. Earlier studies have identified ASIC3 channels, bradykinin receptors, P2X receptors, endoperoxide receptors, and thromboxane receptors as playing a role in evoking the exercise pressor reflex in healthy rats. This review focuses on recent studies using a rat model of PAD in order to determine possible mechanisms contributing to the exaggerated exercise pressor reflex seen in patients with this disease. Twit Text FOAVip The exercise pressor reflex and peripheral artery disease ????Auton Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25458431 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25458431 #FOAvip English Wikipedia <ref>{{Cite pmid|25458431}}</ref> The exercise pressor reflex and peripheral artery disease #MMPMID25458431 Stone AJ; Kaufman MP Auton Neurosci 2015[Mar]; ä (ä): 69-73 PMID25458431show ga The exercise pressor reflex contributes to increases in cardiovascular and ventilatory function during exercise. These reflexive increases are caused by both mechanical and metabolic stimulation of Group III and IV afferents with endings in contracting skeletal muscle. Patients with peripheral artery disease (PAD) have an augmented exercise pressor reflex. Recently, an animal model of PAD was established which allows further investigation of possible mechanisms involved in this augmented reflex. Earlier studies have identified ASIC3 channels, bradykinin receptors, P2X receptors, endoperoxide receptors, and thromboxane receptors as playing a role in evoking the exercise pressor reflex in healthy rats. This review focuses on recent studies using a rat model of PAD in order to determine possible mechanisms contributing to the exaggerated exercise pressor reflex seen in patients with this disease. äThe exercise pressor reflex and peripheral artery disease (epmc).pdf DeepDyve Pubget Overpricing